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Article

Bmi1 is a MYCN target gene that regulates tumorigenesis through repression of KIF1B β and TSLC1 in neuroblastoma

1 Mar 2010

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Share to Ion channel regulation and function
Share to Neuroblastoma Research and Treatments
Share to Signaling Pathways in Disease

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 20190806
DOI: 10.1038/onc.2010.22

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Related research

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Research trail→

Click a neighbor to travelStep 1 · 12 closest

Bmi1 is a MYCN target gene that regulates tumorigenesis through repression of KIF1B β and TSLC1 in neuroblastomaPMID 20190806Read ↗

MYCN and MYC regulate tumor proliferation and tumorigenesis directly through BMI1 in human neuroblastomasSemantic proximity Read ↗

Bmi-1 Is Essential for the Tumorigenicity of Neuroblastoma CellsSemantic proximity Read ↗

BMI ‐1 promotes breast cancer proliferation and metastasis through different mechanisms in different subtypesSemantic proximity Read ↗

Comprehensive analysis and validation reveal potential MYCN regulatory biomarkers associated with neuroblastoma prognosisSemantic proximity Read ↗

BMI1 promotes osteosarcoma proliferation and metastasis by repressing the transcription of SIK1Semantic proximity Read ↗

SMARCE1 promotes neuroblastoma tumorigenesis through assisting MYCN-mediated transcriptional activationSemantic proximity Read ↗

BMI1 as a novel target for drug discovery in cancerSemantic proximity Read ↗

BMI-1 Promotes Ewing Sarcoma Tumorigenicity Independent of <i>CDKN2A</i> RepressionSemantic proximity Read ↗

The transcriptional co-repressor Runx1t1 is essential for MYCN-driven neuroblastoma tumorigenesisSemantic proximity Read ↗

BMI1: A Biomarker of Hematologic MalignanciesSemantic proximity Read ↗

MYCN promotes neuroblastoma malignancy by establishing a regulatory circuit with transcription factor AP4Semantic proximity Read ↗

Metastasis in neuroblastoma: the MYCN questionSemantic proximity Read ↗

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