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Article

Etv4 and Etv5 are required downstream of GDNF and Ret for kidney branching morphogenesis

8 Nov 2009

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Share to Birth, Development, and Health
Share to Genetic and Kidney Cyst Diseases
Share to Renal and related cancers

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 19898483
DOI: 10.1038/ng.476

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

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Etv4 and Etv5 are required downstream of GDNF and Ret for kidney branching morphogenesisPMID 19898483Read ↗

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The molecular control of renal branching morphogenesis: current knowledge and emerging insightsSemantic proximity Read ↗

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E26 transformation-specific transcription variant 5 in development and cancer: modification, regulation and functionSemantic proximity Read ↗

Elf5 is a principal cell lineage specific transcription factor in the kidney that contributes to Aqp 2 and Avpr 2 gene expressionSemantic proximity Read ↗

The contribution of branching morphogenesis to kidney development and diseaseSemantic proximity Read ↗

Six2 and Wnt Regulate Self-Renewal and Commitment of Nephron Progenitors through Shared Gene Regulatory NetworksSemantic proximity Read ↗

Ret and Etv4 Promote Directed Movements of Progenitor Cells during Renal Branching MorphogenesisSemantic proximity Read ↗

Six1 and Six4 are essential for Gdnf expression in the metanephric mesenchyme and ureteric bud formation, while Six1 deficiency alone causes mesonephric-tubule defectsSemantic proximity Read ↗

GDNF is required for kidney development and enteric innervation.Semantic proximity Read ↗

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