Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Multiple Aberrations of Chromosome 3p Detected in Oral Premalignant Lesions

1 Nov 2008

Abstract excerpt

The study of oral premalignant lesions (OPL) is crucial to the identification of initiating genetic events in oral cancer. However, these lesions are minute in size, making it a challenge to recover sufficient DNA from microdissected cells for comprehensive genomic analysis. As a step toward identifying genetic aberrations associated with oral cancer progression, we used tiling-path array comparative genomic...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cholangiocarcinoma and Gallbladder Cancer Studies
Share to Genomic variations and chromosomal abnormalities
Share to Genomics and Chromatin Dynamics

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 19138989
DOI: 10.1158/1940-6207.capr-08-0123

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Multiple Aberrations of Chromosome 3p Detected in Oral Premalignant LesionsPMID 19138989Read ↗

Genomic imbalances in precancerous tissues signal oral cancer risk.Semantic proximity Read ↗

Multiple pathways in the FGF signaling network are frequently deregulated by gene amplification in oral dysplasiasSemantic proximity Read ↗

Concurrent analysis of loss of heterozygosity (LOH) and copy number abnormality (CNA) for oral premalignancy progression using the Affymetrix 10K SNP mapping array.Semantic proximity Read ↗

Loss of Heterozygosity (LOH) Profiles—Validated Risk Predictors for Progression to Oral CancerSemantic proximity Read ↗

Recurrent genomic alterations in sequential progressive leukoplakia and oral cancer: drivers of oral tumorigenesis?Semantic proximity Read ↗

Increased genetic damage in oral leukoplakia from high risk sitesSemantic proximity Read ↗

Recurring DNA copy number gain at chromosome 9p13 plays a role in the activation of multiple candidate oncogenes in progressing oral premalignant lesions.Semantic proximity Read ↗

3p14 and 9p21 loss is a simple tool for predicting second oral malignancy at previously treated oral cancer sites.Semantic proximity Read ↗

Chromosomal Alterations and Gene Expression Changes Associated with the Progression of Leukoplakia to Advanced Gingivobuccal CancerSemantic proximity Read ↗

Allelic imbalance on chromosome 3p in oral dysplastic lesions: an early event in oral carcinogenesis.Semantic proximity Read ↗

Chromosome 3p tumor-suppressor gene alterations in cervical carcinomas.Semantic proximity Read ↗

Comparative genomic hybridization reveals genetic progression of oral squamous cell carcinoma from dysplasia via two different tumourigenic pathwaysSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.