Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Effects of non-selective non-steroidal anti-inflammatory drugs on the aggressiveness of prostate cancer.

The Prostate - 1 Nov 2008

Stock David C, Groome Patti A, Siemens D Robert, Rohland Susan L, Song Zhi

Abstract excerpt

BACKGROUND: Inflammatory mediators have a role in the initiation and progression of prostate cancer. Observed anti-cancer effects of non-steroidal anti-inflammatory drugs (NSAIDs) have consisted largely of those that inhibit inflammatory mechanisms thought to promote an aggressive disease phenotype. Epidemiologic studies have supported a chemopreventive effect but there is little research on a possible protective...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Anti-Inflammatory Agents, Non-Steroidal
Share to Cross-Sectional Studies

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Cytoprotection

Share to Odds Ratio

Share to Prostatic Neoplasms

Share to Registries

Share to Risk Assessment

Share to Survival Analysis

Source

Identifiers

PMID: 18698582
DOI: 10.1002/pros.20834

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026. Manifest publication-graph-cohort-2026-09-05.1.

Source receipt: pubmed, retrieved 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Effects of non-selective non-steroidal anti-inflammatory drugs on the aggressiveness of prostate cancer.PMID 18698582Read ↗

Use of aspirin and other nonsteroidal antiinflammatory medications in relation to prostate cancer risk.Semantic proximity Read ↗

Polymorphisms in genes related to inflammation, NSAID use, and the risk of prostate cancer among Danish men.Semantic proximity Read ↗

Nonsteroidal antiinflammatory drugs and decreased risk of advanced prostate cancer: modification by lymphotoxin alpha.Semantic proximity Read ↗

Up-regulation of the inflammatory-reparative phenotype in human prostate carcinoma.Semantic proximity Read ↗

COX2 genetic variation, NSAIDs, and advanced prostate cancer risk.Semantic proximity Read ↗

Finasteride Inhibits Human Prostate Cancer Cell Invasion through MMP2 and MMP9 DownregulationSemantic proximity Read ↗

Induction of invasive phenotype by Casodex in hormone-sensitive prostate cancer cells.Semantic proximity Read ↗

Chronic nitric oxide exposure induces prostate cell carcinogenesis, involving genetic instability and a pro-tumorigenic secretory phenotype.Semantic proximity Read ↗

Down‐regulated expression of prostasin in high‐grade or hormone‐refractory human prostate cancersSemantic proximity Read ↗

Dietary omega-3 fatty acids, cyclooxygenase-2 genetic variation, and aggressive prostate cancer risk.Semantic proximity Read ↗

Selective growth arrest and phenotypic reversion of prostate cancer cells in vitro by nontoxic pharmacological concentrations of phenylacetate.Semantic proximity Read ↗

Neuroendocrine and immune mediators in prostate cancer progression.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.