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Molecular characterization of acute myeloid leukemia

1 Jan 2008

Abstract excerpt

Acute myeloid leukemia (AML) is a genetically heterogeneous clonal disorder characterized by the accumulation of acquired somatic genetic alterations in hematopoietic progenitor cells that alter normal mechanisms of self-renewal, proliferation and differentiation.[1][1] Non-random clonal chromosome

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PMID: 18591623
DOI: 10.3324/haematol.13345

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