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Article

The role of SLC26A6-mediated chloride/oxalate exchange in causing susceptibility to nephrolithiasis.

The Journal of physiology - 1 Mar 2008

Soleimani Manoocher

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Identifiers

PMID: 18310129
DOI: 10.1113/jphysiol.2007.150565
PMCID: PMC2375665

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The role of SLC26A6-mediated chloride/oxalate exchange in causing susceptibility to nephrolithiasis.PMID 18310129Read ↗

Dominant negative mutation in oxalate transporter SLC26A6 associated with enteric hyperoxaluria and nephrolithiasis.Semantic proximity Read ↗

Structural and functional properties of the transporter SLC26A6 reveal mechanism of coupled anion exchangeSemantic proximity Read ↗

Species differences in Cl- affinity and in electrogenicity of SLC26A6-mediated oxalate/Cl- exchange correlate with the distinct human and mouse susceptibilities to nephrolithiasis.Semantic proximity Read ↗

The SLC26 gene family of anion transporters and channels.Semantic proximity Read ↗

Mutations in SLC26A1 Cause Nephrolithiasis.Semantic proximity Read ↗

Regulated transport of sulfate and oxalate by SLC26A2/DTDST.Semantic proximity Read ↗

Phenotypic and functional analysis of human SLC26A6 variants in patients with familial hyperoxaluria and calcium oxalate nephrolithiasis.Semantic proximity Read ↗

Pathogenetics of the human SLC26 transporters.Semantic proximity Read ↗

Functional comparison of mouse slc26a6 anion exchanger with human SLC26A6 polypeptide variants: differences in anion selectivity, regulation, and electrogenicity.Semantic proximity Read ↗

Type 1 sodium-dependent phosphate transporter acts as a membrane potential-driven urate exporter.Semantic proximity Read ↗

The molecular principles underlying diverse functions of the SLC26 family of proteinsSemantic proximity Read ↗

A Genome-Wide Association Study on Calcium Nephrolithiasis in Chinese Han Population Identifies Novel Susceptible Loci at 4q35.1, 5q31.2 and 18q21.2.Semantic proximity Read ↗

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