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Article

Endothelial nitric oxide synthase polymorphism (-786T->C) and increased risk of angiographic vasospasm after aneurysmal subarachnoid hemorrhage.

Stroke - 1 Apr 2008

Ko Nerissa U, Rajendran Pam, Kim Helen, Rutkowski Martin, Pawlikowska Ludmila, Kwok Pui-Yan, Higashida Randall T, Lawton Michael T, Smith Wade S, Zaroff Jonathan G, Young William L

Abstract excerpt

BACKGROUND AND PURPOSE: Vasospasm after aneurysmal subarachnoid hemorrhage (SAH) remains a leading cause of death and disability after aneurysm rupture. Decreased availability of nitric oxide (NO) may be crucial in its pathogenesis. We hypothesized that endothelial NO synthase (eNOS) polymorphisms may determine susceptibility to vasospasm in SAH patients. METHODS: We conducted a prospective cohort study of SAH...

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Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

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Source

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Identifiers

PMID: 18309169
DOI: 10.1161/STROKEAHA.107.496596
PMCID: PMC2650384

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Source receipt: pubmed, retrieved 7 Sept 2026.

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