Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

ETS transcription factor Erm controls subsynaptic gene expression in skeletal muscles.

Neuron - 6 Sept 2007

Hippenmeyer Simon, Huber Roland M, Ladle David R, Murphy Kenneth, Arber Silvia

Abstract excerpt

Accumulation of specific proteins at synaptic structures is essential for synapse assembly and function, but mechanisms regulating local protein enrichment remain poorly understood. At the neuromuscular junction (NMJ), subsynaptic nuclei underlie motor axon terminals within extrafusal muscle fibers and are transcriptionally distinct from neighboring nuclei. In this study, we show that expression of the ETS...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Down-Regulation
Share to Muscle Weakness
Share to Myasthenia Gravis

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Neuromuscular Junction

Share to Receptors, Nicotinic

Share to Synaptic Membranes

Share to Synaptic Transmission

Share to Transcriptional Activation

Source

Identifiers

PMID: 17785180
DOI: 10.1016/j.neuron.2007.07.028

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

ETS transcription factor Erm controls subsynaptic gene expression in skeletal muscles.PMID 17785180Read ↗

Expression of mutant Ets protein at the neuromuscular synapse causes alterations in morphology and gene expression.Semantic proximity Read ↗

LMO7-null mice exhibit phenotypes consistent with emery-dreifuss muscular dystrophy.Semantic proximity Read ↗

Epigenetic changes as a common trigger of muscle weakness in congenital myopathies.Semantic proximity Read ↗

Impairments in contractility and cytoskeletal organisation cause nuclear defects in nemaline myopathySemantic proximity Read ↗

Structural and functional alterations of neuromuscular synapses in a mouse model of <i>ACTA1</i> congenital myopathySemantic proximity Read ↗

The critical role of membralin in postnatal motor neuron survival and disease.Semantic proximity Read ↗

Genome-wide analysis links emerin to neuromuscular junction activity in Caenorhabditis elegans.Semantic proximity Read ↗

Impairments in contractility and cytoskeletal organisation cause nuclear defects in nemaline myopathySemantic proximity Read ↗

CtBP1 coordinates synaptic, metabolic and contractile changes induced by denervation in skeletal muscleSemantic proximity Read ↗

Distinct regions specify the nuclear membrane targeting of emerin, the responsible protein for Emery-Dreifuss muscular dystrophy.Semantic proximity Read ↗

Acetylcholinesterase Expression in Muscle Is Specifically Controlled by a Promoter-Selective Enhancesome in the First IntronSemantic proximity Read ↗

AML1 is expressed in skeletal muscle and is regulated by innervation.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.