Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Complement 3 is involved in changing the phenotype of human glomerular mesangial cells.

Journal of cellular physiology - 1 Nov 2007

Wan Jian-Xin, Fukuda Noboru, Endo Morito, Tahira Yoshiko, Yao En-Hui, Matsuda Hiroyuki, Ueno Takahiro, Matsumoto Koichi

Abstract excerpt

Complement activation contributes to tissue injury in various forms of glomerulopathy and is characterized by deposition of complement components, which accelerates the progression of chronic renal damage. We recently reported that complement 3 (C3), a critical component of the complement system,...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Biomarkers
Share to Cells, Cultured
Share to Complement C3

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Immunologic Factors

Share to Interferon-gamma

Share to Receptors, Complement

Share to RNA, Small Interfering

Share to Tumor Necrosis Factor-alpha

Source

Identifiers

PMID: 17520688
DOI: 10.1002/jcp.21129

Genes

TNF

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-17520688-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 8 closest

Complement 3 is involved in changing the phenotype of human glomerular mesangial cells.PMID 17520688Read ↗

Role of complement 3a in the synthetic phenotype and angiotensin II-production in vascular smooth muscle cells from spontaneously hypertensive rats.Semantic proximity Read ↗

Complement 3 is involved in the synthetic phenotype and exaggerated growth of vascular smooth muscle cells from spontaneously hypertensive rats.Semantic proximity Read ↗

C3 glomerulonephritis with a severe crescentic phenotype.Semantic proximity Read ↗

S100/Calgranulin-mediated inflammation accelerates left ventricular hypertrophy and aortic valve sclerosis in chronic kidney disease in a receptor for advanced glycation end products-dependent manner.Semantic proximity Read ↗

Humanized C3 Mouse: A Novel Accelerated Model of C3 Glomerulopathy.Semantic proximity Read ↗

Acquisition of the monocyte/macrophage phenotype in human mesangial cells.Semantic proximity Read ↗

Monoclonal antibody against rat podocyte-derived macrophagic cells reacts with crescent-forming cells in an experimental model.Semantic proximity Read ↗

Complement and kidney disease.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.