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Article

Analysis of minK and eNOS genes as candidate loci for predisposition to non-valvular atrial fibrillation.

European heart journal - 1 Jul 2006

Fatini Cinzia, Sticchi Elena, Genuardi Maurizio, Sofi Francesco, Gensini Francesca, Gori Anna Maria, Lenti Meri, Michelucci Antonio, Abbate Rosanna, Gensini Gian Franco

Abstract excerpt

AIM: Mink protein, a beta-subunit of I(ks) potassium channels modulate cardiac cellular electrophysiology, and experimental data demonstrated that NO is involved in cardiac vagal activity and in the inhibition of sympathetic activity. We evaluated the role of eNOS -786T>C, 894G>T, 4a/4b and of minK S38G polymorphisms as predisposing factors to non-valvular atrial fibrillation (NVAF). METHODS AND RESULTS: We...

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Share to Atrial Fibrillation
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Share to Nitric Oxide Synthase Type III

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Share to Polymorphism, Genetic

Share to Polymorphism, Restriction Fragment Length

Share to Potassium Channels, Voltage-Gated

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Identifiers

PMID: 16760206
DOI: 10.1093/eurheartj/ehl087

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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Analysis of minK and eNOS genes as candidate loci for predisposition to non-valvular atrial fibrillation.PMID 16760206Read ↗

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