Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Activation of Kaposi's Sarcoma-Associated Herpesvirus Lytic Gene Expression during Epithelial Differentiation

14 Oct 2005

Abstract excerpt

The oral cavity has been identified as the major site for the shedding of infectious Kaposi's sarcoma-associated herpesvirus (KSHV). While KSHV DNA is frequently detected in the saliva of KSHV seropositive persons, it does not appear to replicate in salivary glands. Some viruses employ the process of epithelial differentiation for productive viral replication. To test if KSHV utilizes the differentiation of oral...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cytomegalovirus and herpesvirus research
Share to Herpesvirus Infections and Treatments
Share to Viral-associated cancers and disorders

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 16227296
DOI: 10.1128/jvi.79.21.13769-13777.2005

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Activation of Kaposi's Sarcoma-Associated Herpesvirus Lytic Gene Expression during Epithelial DifferentiationPMID 16227296Read ↗

Kaposi's Sarcoma-Associated Herpesvirus ORF18 and ORF30 Are Essential for Late Gene Expression during Lytic ReplicationSemantic proximity Read ↗

Differentiated Oral Epithelial Cells Support the HPV Life CycleSemantic proximity Read ↗

Identification of a differentiation-inducible promoter in the E7 open reading frame of human papillomavirus type 16 (HPV-16) in raft cultures of a new cell line containing high copy numbers of episomal HPV-16 DNASemantic proximity Read ↗

Differentiation-induced and constitutive transcription of human papillomavirus type 31b in cell lines containing viral episomesSemantic proximity Read ↗

Orf virus (ORFV) infection in a three-dimensional human skin model: Characteristic cellular alterations and interference with keratinocyte differentiationSemantic proximity Read ↗

Kaposi sarcoma-associated herpesvirus (KSHV) induces a functional tumor-associated phenotype for oral fibroblasts.Semantic proximity Read ↗

Induction of the Human Papillomavirus Type 31 Late Promoter Requires Differentiation but Not DNA AmplificationSemantic proximity Read ↗

In vitro synthesis of oncogenic human papillomaviruses requires episomal genomes for differentiation-dependent late expression.Semantic proximity Read ↗

Human papillomaviruses activate caspases upon epithelial differentiation to induce viral genome amplificationSemantic proximity Read ↗

Human Papillomavirus Type 31b Infection of Human Keratinocytes and the Onset of Early TranscriptionSemantic proximity Read ↗

Temporal Usage of Multiple Promoters during the Life Cycle of Human Papillomavirus Type 31bSemantic proximity Read ↗

HIVtat Alters Epithelial Differentiation State and Increases HPV16 Infectivity in Oral KeratinocytesSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.