Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Transforming growth factor-beta-1 variants are not associated with chronic nonalcoholic pancreatitis.

Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.] - 1 Jan 2005

Böhm Kerstin, Teich Niels, Hoffmeister Albrecht, Mössner Joachim, Keim Volker, Bödeker Hans

Abstract excerpt

BACKGROUND: Pancreatic fibrosis is a key pathological feature of chronic pancreatitis. In vivo and in vitro data have demonstrated that pancreatic stellate cells (PSC) play a central role in pancreatic fibrosis. PSC activation and collagen synthesis are highly controlled by transforming growth factor-beta-1 (TGF-beta1). We evaluated whether functionally relevant genetic variants of TGF-beta1 are associated with...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adolescent
Share to Alleles

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Child, Preschool

Share to Chronic Disease

Share to Pancreatitis

Share to Polymorphism, Genetic

Share to Transforming Growth Factor beta

Source

Identifiers

PMID: 15802940
DOI: 10.1159/000084829

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Transforming growth factor-beta-1 variants are not associated with chronic nonalcoholic pancreatitis.PMID 15802940Read ↗

Transforming growth factor-beta1, interleukin-10 and interferon-gamma cytokine polymorphisms in patients with hereditary, familial and sporadic chronic pancreatitis.Semantic proximity Read ↗

Are genetic variants in the platelet-derived growth factor [beta] gene associated with chronic pancreatitis?Semantic proximity Read ↗

Functional polymorphisms of UDP-glucuronosyltransferases 1A1, 1A6 and 1A8 are not involved in chronic pancreatitis.Semantic proximity Read ↗

The MCP-1 -2518 A/G polymorphism is not a susceptibility factor for chronic pancreatitis.Semantic proximity Read ↗

Analysis of tumor necrosis factor-alpha, transforming growth factor-beta 1, interleukin-10, and interferon-gamma polymorphisms in patients with alcoholic chronic pancreatitis.Semantic proximity Read ↗

Alpha1-antitrypsin genotypes in patients with chronic pancreatitis.Semantic proximity Read ↗

Tumour necrosis factor microsatellite haplotypes are associated with chronic pancreatitis.Semantic proximity Read ↗

Keratin 8 mutations are not associated with familial, sporadic and alcoholic pancreatitis in a population from the United States.Semantic proximity Read ↗

Relevance of transforming growth factor-beta1, interleukin-8, and tumor necrosis factor-alpha polymorphisms in patients with chronic pancreatitis.Semantic proximity Read ↗

Common variants in glyoxalase I do not increase chronic pancreatitis risk.Semantic proximity Read ↗

The functional angiotensin converting enzyme gene I/D polymorphism does not alter susceptibility to chronic pancreatitis.Semantic proximity Read ↗

Influence of cytokine and ICAM-1 gene polymorphisms on susceptibility to chronic pancreatitis.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.