Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Reduced adiponectin and HDL cholesterol without elevated C-reactive protein: Clues to the biology of premature atherosclerosis in Hutchinson-Gilford Progeria Syndrome

1 Mar 2005

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cellular transport and secretion
Share to Cholesterol and Lipid Metabolism
Share to Nuclear Structure and Function

Source

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Identifiers

PMID: 15756215
DOI: 10.1016/j.jpeds.2004.10.064

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Reduced adiponectin and HDL cholesterol without elevated C-reactive protein: Clues to the biology of premature atherosclerosis in Hutchinson-Gilford Progeria SyndromePMID 15756215Read ↗

Hutchinson-gilford progeria syndrome and its relevance to cardiovascular diseases and normal aging.Semantic proximity Read ↗

Hutchinson–Gilford Progeria Syndrome: Cardiovascular Pathologies and Potential TherapiesSemantic proximity Read ↗

HDL and atherosclerosis: Insights from inherited HDL disordersSemantic proximity Read ↗

Metabolic Dysfunction in Hutchinson–Gilford Progeria SyndromeSemantic proximity Read ↗

HDL abnormalities in familial hypercholesterolemia: Focus on biological functionsSemantic proximity Read ↗

Molecular and Cellular Mechanisms Driving Cardiovascular Disease in Hutchinson-Gilford Progeria Syndrome: Lessons Learned from Animal Models.Semantic proximity Read ↗

Phenotype and course of Hutchinson-Gilford progeria syndrome.Semantic proximity Read ↗

Hutchinson-Gilford progeria syndrome: review of the phenotype.Semantic proximity Read ↗

Beyond the genetics of HDL: why is HDL cholesterol inversely related to cardiovascular disease?Semantic proximity Read ↗

Ghrelin delays premature aging in Hutchinson-Gilford progeria syndromeSemantic proximity Read ↗

Normal vascular function despite low levels of high-density lipoprotein cholesterol in carriers of the apolipoprotein A-I(Milano) mutant.Semantic proximity Read ↗

The inverted pattern of circulating miR-221-3p and miR-222-3p associated with isolated low HDL-C phenotype.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.