Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

High-Throughput Retroviral Tagging for Identification of Genes Involved in Initiation and Progression of Mouse Splenic Marginal Zone Lymphomas

1 Jul 2004

Abstract excerpt

Human B-cell lymphomas are frequently associated with specific genetic changes caused by chromosomal translocations that activate proto-oncogenes. For lymphomas of mice expressing murine leukemia virus, mutagenic proviral insertions are thought to play a similar role. Here we report studies designed to determine whether specific retroviral integration sites might be associated with a specific subset of mouse...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Lymphoma Diagnosis and Treatment
Share to T-cell and Retrovirus Studies
Share to Virus-based gene therapy research

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 15231650
DOI: 10.1158/0008-5472.can-03-3885

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

High-Throughput Retroviral Tagging for Identification of Genes Involved in Initiation and Progression of Mouse Splenic Marginal Zone LymphomasPMID 15231650Read ↗

NF-κB deregulation in splenic marginal zone lymphoma.Semantic proximity Read ↗

Genome-based identification of cancer genes by proviral tagging in mouse retrovirus-induced T-cell lymphomas.Semantic proximity Read ↗

Analysis of tumor heterogeneity and cancer gene networks using deep sequencing of MMTV-induced mouse mammary tumors.Semantic proximity Read ↗

KLF2 mutation is the most frequent somatic change in splenic marginal zone lymphoma and identifies a subset with distinct genotype.Semantic proximity Read ↗

Whole-exome sequencing in splenic marginal zone lymphoma reveals mutations in genes involved in marginal zone differentiation.Semantic proximity Read ↗

Large-Scale Identification of Disease Genes Involved in Acute Myeloid LeukemiaSemantic proximity Read ↗

Common sites of retroviral integration in mouse hematopoietic tumors identified by high-throughput, single nucleotide polymorphism-based mapping and bacterial artificial chromosome hybridization.Semantic proximity Read ↗

Insertional mutagenesis in mice deficient for p15Ink4b, p16Ink4a, p21Cip1, and p27Kip1 reveals cancer gene interactions and correlations with tumor phenotypes.Semantic proximity Read ↗

NFAT proteins: emerging roles in cancer progressionSemantic proximity Read ↗

Enhancers Are Major Targets for Murine Leukemia Virus Vector IntegrationSemantic proximity Read ↗

Alteration of BIRC3 and multiple other NF-κB pathway genes in splenic marginal zone lymphomaSemantic proximity Read ↗

Evi-5, a common site of retroviral integration in AKXD T-cell lymphomas, maps near Gfi-1 on mouse chromosome 5Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.