Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Vasoconstrictively acting AT1R A1166C and NOS3 4/5 polymorphisms in recurrent spontaneous abortions (RSA).

American journal of reproductive immunology (New York, N.Y. : 1989) - 1 May 2004

Buchholz Tina, Lohse Peter, Kosian Elke, Thaler Christian J

Abstract excerpt

PROBLEM: Inadequate uteroplacental perfusion is one of the main reasons for recurrent spontaneous abortions (RSA). Coagulation, fibrinolysis, and vasoconstriction affect tissue perfusion. These systems are regulated by different gene products. Polymorphisms can modulate the expression levels of t...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Abortion, Habitual
Share to Homozygote
Share to Leukocytes

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Nitric Oxide Synthase

Share to Nitric Oxide Synthase Type III

Share to Receptor, Angiotensin, Type 2

Share to Tandem Repeat Sequences

Share to Vasoconstriction

Source

Identifiers

PMID: 15212666
DOI: 10.1111/j.1600-0897.2004.00163.x

Genes

NOS3

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 1 Sept 2026. Prepared record publication-15212666-2026-09-01.1. Manifest publication-graph-cohort-2026-09-01.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 1 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Vasoconstrictively acting AT1R A1166C and NOS3 4/5 polymorphisms in recurrent spontaneous abortions (RSA).PMID 15212666Read ↗

Endothelial nitric oxide synthase gene polymorphisms in recurrent spontaneous abortions.Semantic proximity Read ↗

A critical update on endothelial nitric oxide synthase gene variations in women with idiopathic recurrent spontaneous abortion: genetic association study, systematic review and meta-analyses.Semantic proximity Read ↗

Genetic variation in vascular endothelial growth factor gene and its association with recurrent spontaneous abortion.Semantic proximity Read ↗

Polymorphisms in NOS3, ACE and PAI-1 genes and risk of spontaneous recurrent miscarriage in the Gaza Strip.Semantic proximity Read ↗

Polymorphisms in the fibrinolytic pathway genes and the risk of recurrent spontaneous abortion.Semantic proximity Read ↗

The Angiotensin-converting Enzyme Insertion/Deletion Polymorphism as a Common Risk Factor for Major Pregnancy Complications.Semantic proximity Read ↗

Endothelial nitric oxide synthase gene influences the risk of pre-eclampsia, the recurrence of negative pregnancy events, and the maternal-fetal flow.Semantic proximity Read ↗

Association between eNOS gene polymorphisms and the risk of unexplained recurrent spontaneous abortion in Yunnan province, China.Semantic proximity Read ↗

Association of vascular endothelial growth factor gene polymorphisms with recurrent spontaneous abortion in Chinese Han women.Semantic proximity Read ↗

Role of the HLA-G regulatory region polymorphisms in idiopathic recurrent spontaneous abortions (RSA).Semantic proximity Read ↗

Association of Nitric Oxide Synthase and Matrix Metalloprotease Single Nucleotide Polymorphisms with Preeclampsia and Its Complications.Semantic proximity Read ↗

Thrombophilic mutations and polymorphisms, alone or in combination, and recurrent spontaneous abortion.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.