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Article

Foxo Transcription Factors Induce the Atrophy-Related Ubiquitin Ligase Atrogin-1 and Cause Skeletal Muscle Atrophy

1 Apr 2004

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 15109499
DOI: 10.1016/s0092-8674(04)00400-3

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Related research

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Foxo Transcription Factors Induce the Atrophy-Related Ubiquitin Ligase Atrogin-1 and Cause Skeletal Muscle AtrophyPMID 15109499Read ↗

Regulation of autophagy and the ubiquitin–proteasome system by the FoxO transcriptional network during muscle atrophySemantic proximity Read ↗

The initiation factor eIF3‐f is a major target for Atrogin1/MAFbx function in skeletal muscle atrophySemantic proximity Read ↗

The multifaceted impact of physical exercise on FoxO signaling pathwaysSemantic proximity Read ↗

Posttranslational modifications control FoxO3 activity during denervation.Semantic proximity Read ↗

FoxO transcription factors in oxidative stress response and ageing – a new fork on the way to longevity?Semantic proximity Read ↗

FOXO-regulated DEAF1 controls muscle regeneration through autophagySemantic proximity Read ↗

Dynamic FoxO transcription factorsSemantic proximity Read ↗

Regulation of muscle atrophy-related genes by the opposing transcriptional activities of ZEB1/CtBP and FOXO3Semantic proximity Read ↗

FoxO3 Controls Autophagy in Skeletal Muscle In VivoSemantic proximity Read ↗

FoxO1: a novel insight into its molecular mechanisms in the regulation of skeletal muscle differentiation and fiber type specification.Semantic proximity Read ↗

Identification of a KLF5-dependent program and drug development for skeletal muscle atrophySemantic proximity Read ↗

Rapid disuse and denervation atrophy involve transcriptional changes similar to those of muscle wasting during systemic diseasesSemantic proximity Read ↗

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