Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Haplotype analysis of SNAP-25 suggests a role in the aetiology of ADHD.

Molecular psychiatry - 1 Aug 2004

Mill J, Richards S, Knight J, Curran S, Taylor E, Asherson P

Abstract excerpt

Several lines of evidence suggest a role for SNAP-25 (synaptosomal-associated protein of 25 kDa) in the genetic aetiology of ADHD. Most notable is the coloboma mouse mutant, which displays spontaneous hyperactivity and is hemizygous for a deletion spanning this gene. We have screened the SNAP-25 gene using denaturing high-performance liquid chromatography and sequencing, and genotyped six polymorphic...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Attention Deficit Disorder with Hyperactivity
Share to DNA Primers
Share to Exons

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Family

Share to Genetic Markers

Share to Membrane Proteins

Share to Nerve Tissue Proteins

Share to Synaptosomal-Associated Protein 25

Source

Identifiers

PMID: 15007392
DOI: 10.1038/sj.mp.4001482

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Haplotype analysis of SNAP-25 suggests a role in the aetiology of ADHD.PMID 15007392Read ↗

Association study of a SNAP-25 microsatellite and attention deficit hyperactivity disorder.Semantic proximity Read ↗

DNA variation in the SNAP25 gene confers risk to ADHD and is associated with reduced expression in prefrontal cortex.Semantic proximity Read ↗

The Association of SNAP25 Gene Polymorphisms in Attention Deficit/Hyperactivity Disorder: a Systematic Review and Meta-Analysis.Semantic proximity Read ↗

Evidence of association between SNAP25 gene and attention deficit hyperactivity disorder in a Latin American sample.Semantic proximity Read ↗

An association study between SNAP-25 gene and attention-deficit hyperactivity disorder.Semantic proximity Read ↗

Identification of DNA variants in the SNAP-25 gene and linkage study of these polymorphisms and attention-deficit hyperactivity disorder.Semantic proximity Read ↗

Investigation of variation in SNAP-25 and ADHD and relationship to co-morbid major depressive disorder.Semantic proximity Read ↗

DNA pooling analysis of ADHD and genes regulating vesicle release of neurotransmitters.Semantic proximity Read ↗

SNAP-25 gene variations and attention-deficit hyperactivity disorder in Iranian population.Semantic proximity Read ↗

Allelic variants of SNAP25 in a family-based sample of ADHD.Semantic proximity Read ↗

Biased paternal transmission of SNAP-25 risk alleles in attention-deficit hyperactivity disorder.Semantic proximity Read ↗

The SNAP25 gene as a susceptibility gene contributing to attention-deficit hyperactivity disorderSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.