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Article

DSCR1 gene expression is dependent on NFATc1 during cardiac valve formation and colocalizes with anomalous organ development in trisomy 16 mice

12 Dec 2003

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Share to Cardiac Structural Anomalies and Repair
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 14738882
DOI: 10.1016/j.ydbio.2003.10.036

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Related research

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DSCR1 gene expression is dependent on NFATc1 during cardiac valve formation and colocalizes with anomalous organ development in trisomy 16 micePMID 14738882Read ↗

The transcription factor NF-ATc is essential for cardiac valve formationSemantic proximity Read ↗

Nfatc1 Promotes Interstitial Cell Formation During Cardiac Valve Development in ZebrafishSemantic proximity Read ↗

Role of the NF-ATc transcription factor in morphogenesis of cardiac valves and septumSemantic proximity Read ↗

Murine Creld1 controls cardiac development through activation of calcineurin/NFATc1 signaling.Semantic proximity Read ↗

Dscr1, a novel endogenous inhibitor of calcineurin signaling, is expressed in the primitive ventricle of the heart and during neurogenesisSemantic proximity Read ↗

Conserved Transcriptional Regulatory Mechanisms in Aortic Valve Development and DiseaseSemantic proximity Read ↗

Heterozygous missense mutations in NFATC1 are associated with atrioventricular septal defect.Semantic proximity Read ↗

Characterization of Nfatc1 regulation identifies an enhancer required for gene expression that is specific to pro-valve endocardial cells in the developing heart.Semantic proximity Read ↗

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Simultaneous quantitative assessment of two distinct cell lineages with a nuclear-localized dual genetic reporter.Semantic proximity Read ↗

Dishevelled-associated activator of morphogenesis 1 (Daam1) is required for heart morphogenesis.Semantic proximity Read ↗

Nfatc1 Coordinates Valve Endocardial Cell Lineage Development Required for Heart Valve FormationSemantic proximity Read ↗

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