Article
Glutamatergic animal models of schizophrenia.
Annals of the New York Academy of Sciences - 1 Nov 2003
Moghaddam Bita, Jackson Mark E
Abstract excerpt
Several lines of evidence, including recent genetic linkage studies implicating susceptibility genes for schizophrenia, make a strong case that abnormal NMDA receptor-mediated neurotransmission is a major locus for the pathophysiology of schizophrenia. Animal models that are relevant to putative NMDA dysfunction in schizophrenia have excellent face validity for several symptoms of schizophrenia and are important...
Read the complete abstract on PubMedTopics
Share this publication in a Topic to start or enrich a Post.
