Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Absence of transcription factor c-maf causes abnormal terminal differentiation of hypertrophic chondrocytes during endochondral bone development

3 Sept 2003

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Bone Metabolism and Diseases
Share to NF-κB Signaling Pathways
Share to Osteoarthritis Treatment and Mechanisms

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 14512017
DOI: 10.1016/s0012-1606(03)00324-5

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Absence of transcription factor c-maf causes abnormal terminal differentiation of hypertrophic chondrocytes during endochondral bone developmentPMID 14512017Read ↗

Transcriptional control of chondrocyte fate and differentiationSemantic proximity Read ↗

Maturational disturbance of chondrocytes inCbfa1-deficient miceSemantic proximity Read ↗

MEF2C transcription factor controls chondrocyte hypertrophy and bone development.Semantic proximity Read ↗

FoxA Family Members Are Crucial Regulators of the Hypertrophic Chondrocyte Differentiation ProgramSemantic proximity Read ↗

Cbfa1 is a positive regulatory factor in chondrocyte maturation.Semantic proximity Read ↗

Regulation of Tcf7 by Runx2 in chondrocyte maturation and proliferationSemantic proximity Read ↗

Skeletal Malformations Caused by Overexpression of Cbfa1 or Its Dominant Negative Form in ChondrocytesSemantic proximity Read ↗

Significance of MEF2C and RUNX3 Regulation for Endochondral Differentiation of Human Mesenchymal Progenitor CellsSemantic proximity Read ↗

Growth retardation in a mouse model of Kabuki syndrome 2 bears mechanistic similarities to Kabuki syndrome 1Semantic proximity Read ↗

Regulation of Cartilage Development and Diseases by Transcription FactorsSemantic proximity Read ↗

Participation of Cbfa1 in regulation of chondrocyte maturation.Semantic proximity Read ↗

Fate of growth plate hypertrophic chondrocytes: Death or lineage extension?Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.