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Article

Characterization of a preferred site on human chromosome 19q for integration of adeno‐associated virus DNA by non‐homologous recombination.

1 Dec 1992

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Share to Cytomegalovirus and herpesvirus research
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Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 1334463
DOI: 10.1002/j.1460-2075.1992.tb05614.x

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Characterization of a preferred site on human chromosome 19q for integration of adeno‐associated virus DNA by non‐homologous recombination.PMID 1334463Read ↗

Targeted integration of adeno‐associated virus (AAV) into human chromosome 19.Semantic proximity Read ↗

Roles of Adeno-Associated Virus Rep Protein and Human Chromosome 19 in Site-Specific RecombinationSemantic proximity Read ↗

Site-specific integration by adeno-associated virus.Semantic proximity Read ↗

Integration Preferences of Wildtype AAV-2 for Consensus Rep-Binding Sites at Numerous Loci in the Human GenomeSemantic proximity Read ↗

Adeno-associated virus (AAV) site-specific recombination does not require a Rep-dependent origin of replication within the AAV terminal repeatSemantic proximity Read ↗

The recombination signals for adeno-associated virus site-specific integration.Semantic proximity Read ↗

Cellular recombination pathways and viral terminal repeat hairpin structures are sufficient for adeno-associated virus integration in vivo and in vitroSemantic proximity Read ↗

A p5 integration efficiency element mediates Rep-dependent integration into AAVS1 at chromosome 19Semantic proximity Read ↗

Adeno-associated virus (AAV) Rep proteins mediate complex formation between AAV DNA and its integration site in human DNA.Semantic proximity Read ↗

Large-Scale Analysis of Adeno-Associated Virus Vector Integration Sites in Normal Human CellsSemantic proximity Read ↗

Exploiting the adenoassociated virus Rep protein to mediate site-specific integration into the human genome and optimisation of Ad/AAV vector designSemantic proximity Read ↗

A model of oncogenic rearrangements: differences between chromosomal translocation mechanisms and simple double-strand break repairSemantic proximity Read ↗

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