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Article

Repression of TGF-β signaling by the oncogenic protein SKI in human melanomas: consequences for proliferation, survival, and metastasis

19 May 2003

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Share to Cancer-related gene regulation
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 12793438
DOI: 10.1038/sj.onc.1206452

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Related research

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Research trail→

Click a neighbor to travelStep 1 · 12 closest

Repression of TGF-β signaling by the oncogenic protein SKI in human melanomas: consequences for proliferation, survival, and metastasisPMID 12793438Read ↗

Interaction of the Ski Oncoprotein with Smad3 Regulates TGF-β SignalingSemantic proximity Read ↗

Ski and SnoN: negative regulators of TGF-β signalingSemantic proximity Read ↗

A Signal Transduction Pathway from TGF-β1 to SKP2 via Akt1 and c-Myc and its Correlation with Progression in Human MelanomaSemantic proximity Read ↗

Direct Interaction of Ski with Either Smad3 or Smad4 Is Necessary and Sufficient for Ski-mediated Repression of Transforming Growth Factor-β SignalingSemantic proximity Read ↗

The Oncoprotein Ski Acts as an Antagonist of Transforming Growth Factor-β Signaling by Suppressing Smad2 PhosphorylationSemantic proximity Read ↗

TGF-β signaling in tumor suppression and cancer progressionSemantic proximity Read ↗

PI3K signalling is required for a TGFβ-induced epithelial-mesenchymal-like transition (EMT-like) in human melanoma cells.Semantic proximity Read ↗

TGF-β Family Signaling in Tumor Suppression and Cancer Progression.Semantic proximity Read ↗

Alterations in components of the TGF-β superfamily signaling pathways in human cancerSemantic proximity Read ↗

Transforming growth factor-β and the hallmarks of cancer.Semantic proximity Read ↗

SKI and MEL1 Cooperate to Inhibit Transforming Growth Factor-β Signal in Gastric Cancer CellsSemantic proximity Read ↗

The oncoprotein Evi-1 represses TGF-β signalling by inhibiting Smad3Semantic proximity Read ↗

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