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Article

Assessment of the CTNNA3 gene encoding human αT-catenin regarding its involvement in dilated cardiomyopathy

1 Mar 2003

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Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 12596047
DOI: 10.1007/s00439-002-0857-5

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

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Assessment of the CTNNA3 gene encoding human αT-catenin regarding its involvement in dilated cardiomyopathyPMID 12596047Read ↗

Clinical Variability Including Non-Dilated Left Ventricular and Dilated Cardiomyopathy in a Pedigree With an Intragenic CTNNA3 Copy Number Variation.Semantic proximity Read ↗

Expanding the Recessive Spectrum of Dilated Cardiomyopathy: RNA-Level Validation of a Homozygous CTNNA3 Splice-Site Variant.Semantic proximity Read ↗

Alpha-T-catenin is expressed in peripheral nerves as a constituent of Schwann cell adherens junctionsSemantic proximity Read ↗

αT-catenin: A developmentally dispensable, disease-linked member of the α-catenin familySemantic proximity Read ↗

Clinical implications of CTNNA1 germline mutations in asymptomatic carriersSemantic proximity Read ↗

Mutations in the area composita protein αT-catenin are associated with arrhythmogenic right ventricular cardiomyopathySemantic proximity Read ↗

A “human knockout” model to investigate the influence of the α-actinin-3 protein on exercise-induced mitochondrial adaptationsSemantic proximity Read ↗

Loss of αT-catenin alters the hybrid adhering junctions in the heart and leads to dilated cardiomyopathy and ventricular arrhythmia following acute ischemia.Semantic proximity Read ↗

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