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Article

Both the Smad and p38 MAPK pathways play a crucial role in Runx2 expression following induction by transforming growth factor-β and bone morphogenetic protein

9 Oct 2002

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Share to Bone health and treatments
Share to Bone Metabolism and Diseases
Share to TGF-β signaling in diseases

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 12370805
DOI: 10.1038/sj.onc.1205937

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Both the Smad and p38 MAPK pathways play a crucial role in Runx2 expression following induction by transforming growth factor-β and bone morphogenetic proteinPMID 12370805Read ↗

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Smad3 and Smad4 cooperate with c-Jun/c-Fos to mediate TGF-β-induced transcriptionSemantic proximity Read ↗

SMADs: mediators and regulators of TGF-β signalingSemantic proximity Read ↗

TGF‐β receptor‐mediated signalling through Smad2, Smad3 and Smad4Semantic proximity Read ↗

Transcriptional Regulatory Cascades in Runx2-Dependent Bone DevelopmentSemantic proximity Read ↗

Role and regulation of RUNX2 in osteogenesisSemantic proximity Read ↗

Smad3/AP-1 interactions control transcriptional responses to TGF-β in a promoter-specific mannerSemantic proximity Read ↗

p38 MAPK Signaling in Osteoblast DifferentiationSemantic proximity Read ↗

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