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Article

Insights from mouse models of absence epilepsy into Ca2+ channel physiology and disease etiology.

Cellular and molecular neurobiology - 1 Apr 2002

Felix Ricardo

Abstract excerpt

1. Changes in intracellular Ca2+ ([Ca2+]i) levels provide signals that allow neurons to respond to a host of external stimuli. A major mechanism for elevating [Ca2+]i is the influx of extracellular Ca2+ through voltage-gated channels (Ca(V)) in the plasma membrane. Malfunction in Ca(V) due to mutations in genes encoding channel proteins are increasingly being implicated in causing disease conditions, termed...

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Source

Identifiers

PMID: 12363194
DOI: 10.1023/a:1019807719343

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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