Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The unique ultrastructure of secretory membranes in gastric parietal cells depends upon the presence of H+, K+ -ATPase.

Cell and tissue research - 1 Sept 2002

Miller Marian L, Judd Louise M, Van Driel Ian R, Andringa Anastasia, Flagella Michael, Bell Sheila M, Schultheis Patrick J, Spicer Zachary, Shull Gary E

Abstract excerpt

Ion transporters play a central role in gastric acid secretion. To determine whether some of these transporters are necessary for the normal ultrastructure of secretory membranes in gastric parietal cells, mice lacking transporters for H+, K+, Cl-, and Na+ were examined for alterations in volume...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Intracellular Membranes
Share to Mice
Share to Mice, Knockout

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Microscopy, Electron

Share to Microvilli

Share to Protein Subunits

Share to Secretory Vesicles

Share to Sodium-Hydrogen Exchangers

Source

Identifiers

PMID: 12195293
DOI: 10.1007/s00441-002-0606-z

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 1 Sept 2026. Prepared record publication-12195293-2026-09-01.1. Manifest publication-graph-cohort-2026-09-01.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 1 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The unique ultrastructure of secretory membranes in gastric parietal cells depends upon the presence of H+, K+ -ATPase.PMID 12195293Read ↗

Impaired gastric acid secretion in mice with a targeted disruption of the NHE4 Na+/H+ exchanger.Semantic proximity Read ↗

Mice with a targeted disruption of the AE2 Cl-/HCO3- exchanger are achlorhydric.Semantic proximity Read ↗

Regulation of gastric epithelial cell development revealed in H(+)/K(+)-ATPase beta-subunit- and gastrin-deficient mice.Semantic proximity Read ↗

Phosphorylation of ezrin on threonine 567 produces a change in secretory phenotype and repolarizes the gastric parietal cell.Semantic proximity Read ↗

Helicobacter pylori VacA Disrupts Apical Membrane-Cytoskeletal Interactions in Gastric Parietal CellsSemantic proximity Read ↗

Physiological relevance of cell-specific distribution patterns of CFTR, NKCC1, NBCe1, and NHE3 along the crypt-villus axis in the intestineSemantic proximity Read ↗

Molecular Regulation of Na+‐K+‐2C1‐ Cotransporter (NKCC1) and Epithelial Chloride SecretionSemantic proximity Read ↗

Knockout mouse models for intestinal electrolyte transporters and regulatory PDZ adaptors: new insights into cystic fibrosis, secretory diarrhoea and fructose‐induced hypertensionSemantic proximity Read ↗

Mice lacking the basolateral Na-K-2Cl cotransporter have impaired epithelial chloride secretion and are profoundly deaf.Semantic proximity Read ↗

[Localization, ion transport activity, and physiological function of mammalian organellar NHEs].Semantic proximity Read ↗

Ion transport in the intestineSemantic proximity Read ↗

Abnormal Rab11-Rab8-vesicles cluster in enterocytes of patients with microvillus inclusion disease.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.