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Article

Early embryonic lethality in mice deficient in the p110β catalytic subunit of PI 3-kinase

1 Mar 2002

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 11919689
DOI: 10.1007/bf02684023

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Early embryonic lethality in mice deficient in the p110β catalytic subunit of PI 3-kinasePMID 11919689Read ↗

Early embryonic lethality in mice deficient in the p110β catalytic subunit of PI 3-kinaseSemantic proximity Read ↗

Critical role for the p110alpha phosphoinositide-3-OH kinase in growth and metabolic regulation.Semantic proximity Read ↗

Proliferative defect and embryonic lethality in mice homozygous for a deletion in the p110alpha subunit of phosphoinositide 3-kinase.Semantic proximity Read ↗

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Hypoglycaemia, liver necrosis and perinatal death in mice lacking all isoforms of phosphoinositide 3-kinase p85 alpha.Semantic proximity Read ↗

The p110α isoform of PI3K is essential for proper growth factor signaling and oncogenic transformationSemantic proximity Read ↗

Signalling by PI3K isoforms: insights from gene-targeted mice.Semantic proximity Read ↗

Both p110α and p110β isoforms of PI3K can modulate the impact of loss-of-function of the PTEN tumour suppressorSemantic proximity Read ↗

Mouse Models for Exploring the Biological Consequences and Clinical Significance of PIK3CA Mutations.Semantic proximity Read ↗

p110δ PI3 kinase pathway: emerging roles in cancerSemantic proximity Read ↗

Agammaglobulinemia and absent B lineage cells in a patient lacking the p85α subunit of PI3KSemantic proximity Read ↗

Mouse models of PI(3,5)P2 deficiency with impaired lysosome function.Semantic proximity Read ↗

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