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Article

LMP2A survival and developmental signals are transmitted through Btk-dependent and Btk-independent pathways.

Virology - 5 Dec 2001

Merchant M, Longnecker R

Abstract excerpt

The latent membrane protein 2A (LMP2A) of Epstein--Barr virus (EBV) has been implicated in controlling viral latency due to the ability of LMP2A to block B cell antigen receptor (BCR) signaling in vitro and to alter B cell development and enhance B cell survival in vivo. These LMP2A functions require interactions with the protein tyrosine kinases Syk and Lyn. However, a role for the Bruton's tyrosine kinase (Btk)...

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Source

Identifiers

PMID: 11878875
DOI: 10.1006/viro.2001.1187

Genes

BCR

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

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