Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

A mutation in the AMPA-type glutamate receptor, glr-1, blocks olfactory associative and nonassociative learning in Caenorhabditis elegans.

Behavioral neuroscience - 1 Jun 2001

Morrison G E, van der Kooy D

Abstract excerpt

The alpha-amino-3-hydroxy-5-methyl-4-isoxazole propionate (AMPA)-type ionotropic glutamate receptor mediates fast excitatory neurotransmission in the vertebrate brain and is important for synaptic plasticity and the initial induction of long-term potentiation (LTP). This study found that the putative Caenorhabditis elegans AMPA receptor gene, glr-1, plays a significant role in experience-dependent behavior in C....

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Association Learning
Share to Caenorhabditis elegans Proteins

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Chemoreceptor Cells

Share to Conditioning, Classical

Share to Long-Term Potentiation

Share to Receptors, AMPA

Share to Receptors, Glutamate

Source

Identifiers

PMID: 11439453
DOI: 10.1037//0735-7044.115.3.640

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

A mutation in the AMPA-type glutamate receptor, glr-1, blocks olfactory associative and nonassociative learning in Caenorhabditis elegans.PMID 11439453Read ↗

Olfactory associative learning in Caenorhabditis elegans is impaired in lrn-1 and lrn-2 mutants.Semantic proximity Read ↗

Genetic dissection of memory for associative and non-associative learning in Caenorhabditis elegans.Semantic proximity Read ↗

Ubiquitin and AP180 regulate the abundance of GLR-1 glutamate receptors at postsynaptic elements in C. elegans.Semantic proximity Read ↗

GLR-1, a non-NMDA glutamate receptor homolog, is critical for long-term memory in Caenorhabditis elegans.Semantic proximity Read ↗

Synaptic code for sensory modalities revealed by C. elegans GLR-1 glutamate receptor.Semantic proximity Read ↗

Memory in Caenorhabditis elegans is mediated by NMDA-type ionotropic glutamate receptors.Semantic proximity Read ↗

Dopamine modulation of avoidance behavior in Caenorhabditis elegans requires the NMDA receptor NMR-1.Semantic proximity Read ↗

GRLD-1 regulates cell-wide abundance of glutamate receptor through post-transcriptional regulation.Semantic proximity Read ↗

[Molecular genetics on behavioral plasticity in Caenorhabditis elegans: mechanisms for associative learning].Semantic proximity Read ↗

A conserved function of C. elegans CASY-1 calsyntenin in associative learning.Semantic proximity Read ↗

Neuronal control of locomotion in C. elegans is modified by a dominant mutation in the GLR-1 ionotropic glutamate receptor.Semantic proximity Read ↗

Mutations that prevent associative learning in C. elegans.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.