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Article

Bone Dysplasia Sclerosteosis Results from Loss of the SOST Gene Product, a Novel Cystine Knot–Containing Protein

1 Mar 2001

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 11179006
DOI: 10.1086/318811

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Related research

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Bone Dysplasia Sclerosteosis Results from Loss of the SOST Gene Product, a Novel Cystine Knot–Containing ProteinPMID 11179006Read ↗

Increased bone density in sclerosteosis is due to the deficiency of a novel secreted protein (SOST)Semantic proximity Read ↗

Genetics of Sost/SOST in sclerosteosis and van Buchem disease animal modelsSemantic proximity Read ↗

Role of Sostdc1 in skeletal biology and cancerSemantic proximity Read ↗

A Novel Loss-of-Sclerostin Function Mutation in a First Egyptian Family with Sclerosteosis.Semantic proximity Read ↗

Sclerosteosis caused by a novel nonsense mutation of SOST in a consanguineous family.Semantic proximity Read ↗

SOST/sclerostin, an osteocyte-derived negative regulator of bone formationSemantic proximity Read ↗

A Novel Mutation in a Gene Causes Sclerosteosis in a Family of Mediterranean Origin.Semantic proximity Read ↗

Novel SOST gene mutation in a sclerosteosis patient and her parents.Semantic proximity Read ↗

Sclerosing bone dysplasias: genetic, clinical and radiology update of hereditary and non-hereditary disorders.Semantic proximity Read ↗

Molecular and radiological diagnosis of sclerosing bone dysplasiasSemantic proximity Read ↗

Targeted deletion of the sclerostin gene in mice results in increased bone formation and bone strength.Semantic proximity Read ↗

Patients with sclerosteosis and disease carriers: Human models of the effect of sclerostin on bone turnoverSemantic proximity Read ↗

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