Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Interdependence of environmental factors influencing reciprocal patterns of gene expression in virulent <i>Borrelia burgdorferi</i>

1 Sept 2000

Abstract excerpt

The paradigm for differential antigen expression in Borrelia burgdorferi, the agent of Lyme disease, is the reciprocal expression of its outer surface (lipo)proteins (Osp) A and C; as B. burgdorferi transitions from its arthropod vector into mammalian tissue, ospC is upregulated, and ospA is downregulated. In the current study, using B. burgdorferi cultivated under varying conditions in BSK-H medium, we found...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Insect and Pesticide Research
Share to Vector-borne infectious diseases
Share to Viral Infections and Vectors

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 10998177
DOI: 10.1046/j.1365-2958.2000.02104.x

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Interdependence of environmental factors influencing reciprocal patterns of gene expression in virulent <i>Borrelia burgdorferi</i>PMID 10998177Read ↗

Analysis of the RpoS regulon in <i>Borrelia burgdorferi</i> in response to mammalian host signals provides insight into RpoS function during the enzootic cycleSemantic proximity Read ↗

Expression of <i>Borrelia burgdorferi</i> OspC and DbpA is controlled by a RpoN–RpoS regulatory pathwaySemantic proximity Read ↗

Borrelia burgdorferiin Tick Cell Culture Modulates Expression of Outer Surface Proteins A and C in Response to TemperatureSemantic proximity Read ↗

<i><scp>B</scp>orrelia burgdorferi</i> oxidative stress regulator <scp>BosR</scp> directly represses lipoproteins primarily expressed in the tick during mammalian infectionSemantic proximity Read ↗

Alternate Sigma Factor RpoS Is Required for the In Vivo-Specific Repression of<i>Borrelia burgdorferi</i>Plasmid lp54-Borne<i>ospA</i>and<i>lp6.6</i>GenesSemantic proximity Read ↗

Adaptation of<i>Borrelia burgdorferi</i>in the tick and the mammalian hostSemantic proximity Read ↗

The Lyme disease spirochete, Borrelia burgdorferi, as a model vector-borne pathogen: insights on regulation of gene and protein expressionSemantic proximity Read ↗

Role of the Surface Lipoprotein BBA07 in the Enzootic Cycle of<i>Borrelia burgdorferi</i>Semantic proximity Read ↗

Activation of the RpoN-RpoS regulatory pathway during the enzootic life cycle of Borrelia burgdorferiSemantic proximity Read ↗

RpoS is not central to the general stress response in Borrelia burgdorferi but does control expression of one or more essential virulence determinants.Semantic proximity Read ↗

DNA microarray analysis of differential gene expression in <i>Borrelia burgdorferi</i> , the Lyme disease spirocheteSemantic proximity Read ↗

Regulation of OspE-Related, OspF-Related, and Elp Lipoproteins of Borrelia burgdorferi Strain 297 by Mammalian Host-Specific SignalsSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.