Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Chromosomal alterations in human pancreatic endocrine tumors

1 Sept 2000

Abstract excerpt

Comparative genomic hybridization (CGH) was used to investigate changes in DNA copy numbers in 25 paraffin-embedded samples of pancreatic endocrine tumors from 23 patients. Insulin was the dominant hormone in 12, glucagon in 7, somatostatin in 1, and pancreatic polypeptide in 2 tumors. One to 15 (mean, 8.1) changes in DNA copy numbers were observed in 22 of the 25 tumors. The most recurrent aberration, found in...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Neuroendocrine Tumor Research Advances
Share to Pancreatic and Hepatic Oncology Research
Share to Pancreatic function and diabetes

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 10918398
DOI: 10.1002/1098-2264(2000)9999:9999<::aid-gcc1011>3.0.co;2-z

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Chromosomal alterations in human pancreatic endocrine tumorsPMID 10918398Read ↗

Comparative genomic hybridization reveals frequent gains of 20q, 8q, 11q, 12p, and 17q, and losses of 18q, 9p, and 15q in pancreatic cancerSemantic proximity Read ↗

Frequent gain of copy number on the long arm of chromosome 20 in human pancreatic adenocarcinomaSemantic proximity Read ↗

Analysis of sporadic neuroendocrine tumours of the enteropancreatic system by comparative genomic hybridisationSemantic proximity Read ↗

Genome-Wide Array-Based Comparative Genomic Hybridization Reveals Multiple Amplification Targets and Novel Homozygous Deletions in Pancreatic Carcinoma Cell LinesSemantic proximity Read ↗

Genomic imbalances in the progression of endocrine pancreatic tumorsSemantic proximity Read ↗

Frequent amplification of 8q24, 11q, 17q, and 20q‐specific genes in pancreatic cancerSemantic proximity Read ↗

Allelic alterations in pancreatic endocrine tumors identified by genome-wide single nucleotide polymorphism analysis.Semantic proximity Read ↗

Cytogenetic analysis of pancreatic carcinomas: Intratumor heterogeneity and nonrandom pattern of chromosome aberrationsSemantic proximity Read ↗

Genomic alterations in distal bile duct carcinoma by comparative genomic hybridization and karyotype analysisSemantic proximity Read ↗

Sex chromosome anomalies in pancreatic endocrine tumorsSemantic proximity Read ↗

Frequent genomic imbalances in chromosomes 17, 19, and 22q in peripheral nerve sheath tumours detected by comparative genomic hybridization analysisSemantic proximity Read ↗

Comparison of mutations of ras oncogene in human pancreatic exocrine and endocrine tumors.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.