Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Comparative genomic hybridization reveals frequent gains of 20q, 8q, 11q, 12p, and 17q, and losses of 18q, 9p, and 15q in pancreatic cancer

1 Dec 1997

Abstract excerpt

Comparative genomic hybridization (CGH) was used to screen for genomic imbalances in 24 exocrine pancreatic carcinomas, including II low-passage cell lines (4-8 subcultures) and 13 uncultured samples. Aberrations were found in all cell lines and in seven of the 13 biopsies. The most frequent changes in the cell lines were gains of 20q (91%), 11q (64%), 17q (64%), 19q (64%), 8q, 12p, 14q, and 20p (55%), and losses...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer Genomics and Diagnostics
Share to Cholangiocarcinoma and Gallbladder Cancer Studies
Share to Pancreatic and Hepatic Oncology Research

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 9408755
DOI: 10.1002/(sici)1098-2264(199712)20:4<383::aid-gcc10>3.0.co;2-o

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Comparative genomic hybridization reveals frequent gains of 20q, 8q, 11q, 12p, and 17q, and losses of 18q, 9p, and 15q in pancreatic cancerPMID 9408755Read ↗

Frequent gain of copy number on the long arm of chromosome 20 in human pancreatic adenocarcinomaSemantic proximity Read ↗

Frequent amplification of 8q24, 11q, 17q, and 20q‐specific genes in pancreatic cancerSemantic proximity Read ↗

Chromosomal alterations in human pancreatic endocrine tumorsSemantic proximity Read ↗

Comparative genomic hybridization of esophageal and gastroesophageal adenocarcinomas shows consensus areas of DNA gain and lossSemantic proximity Read ↗

Genome-Wide Array-Based Comparative Genomic Hybridization Reveals Multiple Amplification Targets and Novel Homozygous Deletions in Pancreatic Carcinoma Cell LinesSemantic proximity Read ↗

Cytogenetic analysis of pancreatic carcinomas: Intratumor heterogeneity and nonrandom pattern of chromosome aberrationsSemantic proximity Read ↗

Pancreatic acinar carcinoma shows a distinct pattern of chromosomal imbalances by comparative genomic hybridization.Semantic proximity Read ↗

<i>Intersex-like</i> ( <i>IXL</i> ) Is a Cell Survival Regulator in Pancreatic Cancer with 19q13 AmplificationSemantic proximity Read ↗

Association of poor prognosis with loss of 12q, 17p, and 18q, and concordant loss of 6q/17p and 12q/18q in human pancreatic ductal adenocarcinoma.Semantic proximity Read ↗

The structural basis of molecular genetic deletions. An integration of classical cytogenetic and molecular analyses in pancreatic adenocarcinoma.Semantic proximity Read ↗

Genomic alterations in distal bile duct carcinoma by comparative genomic hybridization and karyotype analysisSemantic proximity Read ↗

Genomic imbalances in the progression of endocrine pancreatic tumorsSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.