Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Lack of association of angiotensin-converting enzyme and angiotensinogen genes polymorphisms with left ventricular structure in young normotensive men.

Blood pressure - 1 Jan 2000

Linhart A, Sedlácek K, Jáchymová M, Jindra A, Beran S, Vondrácek V, Heller S, Horký K

Abstract excerpt

Left ventricular (LV) hypertrophy is a strong predictive factor for cardiovascular morbidity and mortality. LV structure and function are influenced by many variables, including genetic background. The potential role of gene polymorphisms of different components of the renin angiotensin system remains controversial. The aim of this study was to determine the influence of deletion/insertion (D/I) polymorphism of...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Angiotensinogen
Share to DNA Transposable Elements

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Echocardiography

Share to Gene Deletion

Share to Heart Ventricles

Share to Peptidyl-Dipeptidase A

Share to Polymorphism, Genetic

Share to Reference Values

Source

Identifiers

PMID: 10854008

Genes

ACE

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Lack of association of angiotensin-converting enzyme and angiotensinogen genes polymorphisms with left ventricular structure in young normotensive men.PMID 10854008Read ↗

Lack of association between ACE gene polymorphism and left ventricular hypertrophy in essential hypertension.Semantic proximity Read ↗

Angiotensin-converting enzyme polymorphism in healthy young subjects: relationship to left ventricular mass and functions.Semantic proximity Read ↗

Left ventricular size, mass, and function in relation to angiotensin-converting enzyme gene polymorphism in humans.Semantic proximity Read ↗

Usefulness of the I/D angiotensin-converting enzyme genotype for detecting the risk of left ventricular hypertrophy in pharmacologically treated hypertensive men.Semantic proximity Read ↗

Lack of association of the renin-angiotensin system genes polymorphisms and left ventricular hypertrophy in hypertension.Semantic proximity Read ↗

Deletion polymorphism of the angiotensin-converting enzyme gene is independently associated with left ventricular mass and geometric remodeling in systemic hypertension.Semantic proximity Read ↗

Absence of Association or Genetic Linkage between the Angiotensin-Converting–Enzyme Gene and Left Ventricular MassSemantic proximity Read ↗

Absence of association or genetic linkage between the angiotensin-converting-enzyme gene and left ventricular mass.Semantic proximity Read ↗

Angiotensinogen M235T polymorphism and left ventricular indices in treated hypertensive patients with normal coronary arteries.Semantic proximity Read ↗

ACE gene polymorphism and cardiac structure in patients with insulin resistance.Semantic proximity Read ↗

[I/D gene polymorphism of the angiotensin-converting enzyme and left ventricular hypertrophy. Response to converting enzyme inhibitors].Semantic proximity Read ↗

Hypertensive left ventricular remodeling and ACE-gene polymorphism.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.