Article
Fibroblast growth factor receptor-4 splice variants cause deletion of a critical tyrosine.
IUBMB life - 1 Jul 1999
van Heumen W R, Claxton C, Pickles J O
Abstract excerpt
We have identified two novel isoforms of fibroblast growth factor receptor-4 (FGFR4). They result from alternative splicing of intron 17. Two transcripts, both slightly larger than the one coding for the known mouse FGFR4, are generated. The shortest (FGFR4-17a) includes the 31-most 3'-nucleotides of intron 17; the longest (FGFR4-17b) includes all 114 nucleotides of intron 17. Translation of the FGFR4-17a and...
Read the complete abstract on PubMedTopics
Share this publication in a Topic to start or enrich a Post.
