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Article

Telomere shortening during aging of human osteoblasts in vitro and leukocytes in vivo: lack of excessive telomere loss in osteoporotic patients

1 Jan 1999

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Share to Telomeres, Telomerase, and Senescence

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 10100154
DOI: 10.1016/s0047-6374(98)00114-6

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Telomere shortening during aging of human osteoblasts in vitro and leukocytes in vivo: lack of excessive telomere loss in osteoporotic patientsPMID 10100154Read ↗

Reconstituting Telomerase Activity Using the Telomerase Catalytic Subunit Prevents the Telomere Shorting and Replicative Senescence in Human OsteoblastsSemantic proximity Read ↗

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Senescence‐associated intrinsic mechanisms of osteoblast dysfunctionsSemantic proximity Read ↗

Defects in telomere maintenance molecules impair osteoblast differentiation and promote osteoporosisSemantic proximity Read ↗

Replicative Aging and Gene Expression in Long-Term Cultures of Human Bone Marrow Stromal CellsSemantic proximity Read ↗

Telomere length and telomerase activity during expansion and differentiation of human mesenchymal stem cells and chondrocytesSemantic proximity Read ↗

The relationship between ultra-short telomeres, aging of articular cartilage and the development of human hip osteoarthritisSemantic proximity Read ↗

Telomerase-deficient mice exhibit bone loss owing to defects in osteoblasts and increased osteoclastogenesis by inflammatory microenvironment.Semantic proximity Read ↗

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