Back to search

Article

Immune-Mediated Coronary Stent Failure: From PCI-Induced Vascular Injury to In-Stent Restenosis, Neoatherosclerosis and Stent Thrombosis

2026-06-25

Abstract excerpt

Percutaneous coronary intervention (PCI) treats focal coronary obstruction by compressing plaque, injuring the vessel wall and placing a metallic or bioresorbable scaffold. Most treated segments heal, but a small minority enter a prolonged, excessive or unstable repair state that contributes to in-stent restenosis (ISR), in-stent neoatherosclerosis and stent thrombosis (ST). Human evidence is strongest for delayed...

Topics

Open a Topic to create a Post that cites this publication.

Identifiers and source

Literature Corpus work
7cab122a-a7c1-546f-bf9b-3c7abb979229
DOI
10.20944/preprints202606.1930.v1
Open publication

Related research

Semantic proximity does not establish scientific evidence.

Click a neighbor to travelStep 1 · 12 closest
Interactive article relationship graphSelect a related publication card to move it into the centre and load its closest explainable connections. Solid lines are source-backed structured connections. Dashed lines are semantic discovery signals and are not scientific evidence.
Immune-Mediated Coronary Stent Failure: From PCI-Induced Vascular Injury to In-Stent Restenosis, Neoatherosclerosis and Stent ThrombosisDOI 10.20944/preprints202606.1930.v1
In-stent restenosis is associated with proliferative skin healing and specific immune and endothelial cell profiles: results from the RACHEL trial.Semantic proximity
Innovations of Diagnosis and Treatment of Coronary Artery DiseaseSemantic proximity
Atherosclerosis as a disease of failed endogenous repairSemantic proximity
Biological responses in stented arteries.Semantic proximity
Inflammatory Phenotype by OCT Coronary Imaging: Specific Features Among De Novo Lesions, In-Stent Neointima, and In-Stent Neo-Atherosclerosis.Semantic proximity
Microparticles from ISR patients participate in vascular intimal hyperplasia by promoting oxidative stress, inflammation and adhesion through ERK and P38 pathwaysSemantic proximity
A decline in platelet activation and inflammatory cell infiltration is associated with the phenotypic redifferentiation of neointimal smooth muscle cells after bare-metal stent implantation in acute coronary syndrome.Semantic proximity
The Microvascular–Immune Interface in Cardiovascular Disease: A Stage-Based Framework of Microvascular FailureSemantic proximity
Silent Myocardial Infarction Revisited: Immuno-metabolic Mechanisms, Multimodal Biomarkers, and Translational Diagnostics.Semantic proximity
Plaque erosion and acute coronary syndromes: phenotype, molecular characteristics and future directions.Semantic proximity
Innate immune cells in ischaemic heart disease: does myocardial infarction beget myocardial infarction?Semantic proximity
A systematic review and bioinformatic study on clinical, paraclinical, and genetic factors predisposing to stent restenosis following percutaneous coronary intervention.Semantic proximity
Select a neighboring publication to make it the new centre.