Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Increased p16 expression with first senescence arrest in human mammary epithelial cells and extended growth capacity with p16 inactivation

16 Jul 1998

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Advanced Breast Cancer Therapies
Share to Cancer Immunotherapy and Biomarkers
Share to Cancer-related Molecular Pathways

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 9674704
DOI: 10.1038/sj.onc.1201919

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Increased p16 expression with first senescence arrest in human mammary epithelial cells and extended growth capacity with p16 inactivationPMID 9674704Read ↗

Reversal of human cellular senescence: roles of the p53 and p16 pathwaysSemantic proximity Read ↗

Normal human mammary epithelial cells spontaneously escape senescence and acquire genomic changesSemantic proximity Read ↗

Molecular changes accompanying senescence and immortalization of cultured human mammary epithelial cellsSemantic proximity Read ↗

The specific role of pRb in p16<sup>INK4A</sup>-mediated arrest of normal and malignant human breast cellsSemantic proximity Read ↗

Suppression of p16 alleviates the senescence-associated secretory phenotypeSemantic proximity Read ↗

p16INK4asuppresses BRCA1-deficient mammary tumorigenesisSemantic proximity Read ↗

p16High senescence restricts cellular plasticity during somatic cell reprogrammingSemantic proximity Read ↗

Early growth response 2 (EGR2) is a novel regulator of the senescence programSemantic proximity Read ↗

Raf-1-induced growth arrest in human mammary epithelial cells is p16-independent and is overcome in immortal cells during conversionSemantic proximity Read ↗

Suppression of p16 alleviates the senescence-associated secretory phenotypeSemantic proximity Read ↗

Tumor suppressor p16 <sup>INK4A</sup> is necessary for survival of cervical carcinoma cell linesSemantic proximity Read ↗

Co-Regulation of p16INK4a and Migratory Genes in Culture Conditions that Lead to Premature Senescence in Human KeratinocytesSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.