Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Reduced transmitter release conferred by mutations in the slowpoke-encoded Ca2(+)-activated K+ channel gene of Drosophila.

Invertebrate neuroscience : IN - 1 Jun 1996

Warbington L, Hillman T, Adams C, Stern M

Abstract excerpt

Potassium channels control the repolarization of nerve terminals and thus play important roles in the control of synaptic transmission. Here we describe the effects of mutations in the slowpoke gene, which is the structural gene for a calcium activated potassium channel, on transmitter release at the neuromuscular junction in Drosophila melanogaster. Surprisingly, we find that the slowpoke mutant exhibits reduced...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Calcium
Share to Drosophila melanogaster
Share to Drosophila Proteins

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Electrophysiology

Share to Genes, Insect

Share to Mutation

Share to Osmolar Concentration

Share to Potassium Channels

Source

Identifiers

PMID: 9372155
DOI: 10.1007/BF02336660

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Reduced transmitter release conferred by mutations in the slowpoke-encoded Ca2(+)-activated K+ channel gene of Drosophila.PMID 9372155Read ↗

Potassium channels in Drosophila: historical breakthroughs, significance, and perspectives.Semantic proximity Read ↗

Genetic modification of potassium channels in Drosophila Shaker mutants.Semantic proximity Read ↗

Dissection of synaptic excitability phenotypes by using a dominant-negative Shaker K+ channel subunit.Semantic proximity Read ↗

Unmasking of a novel potassium current in Drosophila by a mutation and drugs.Semantic proximity Read ↗

Distinct frequency-dependent regulation of nerve terminal excitability and synaptic transmission by IA and IK potassium channels revealed by Drosophila Shaker and Shab mutations.Semantic proximity Read ↗

In vivo analysis of a gain-of-function mutation in the Drosophila eag-encoded K+ channel.Semantic proximity Read ↗

Molecular separation of two behavioral phenotypes by a mutation affecting the promoters of a Ca-activated K channel.Semantic proximity Read ↗

Genetic and pharmacological identification of ion channels central to the Drosophila cardiac pacemaker.Semantic proximity Read ↗

Distinct roles of Drosophila cacophony and Dmca1D Ca(2+) channels in synaptic homeostasis: genetic interactions with slowpoke Ca(2+) -activated BK channels in presynaptic excitability and postsynaptic response.Semantic proximity Read ↗

Complementation of physiological and behavioral defects by a slowpoke Ca(2+) -activated K(+) channel transgene.Semantic proximity Read ↗

Identification and characterization of inebriated, a gene affecting neuronal excitability in Drosophila.Semantic proximity Read ↗

Synaptic excitation is regulated by the postsynaptic dSK channel at the Drosophila larval NMJ.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.