Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

sfi-independent filamentation in Escherichia coli Is lexA dependent and requires DNA damage for induction.

Journal of bacteriology - 1 Mar 1997

Hill T M, Sharma B, Valjavec-Gratian M, Smith J

Abstract excerpt

In Escherichia coli, damage to DNA induces the expression of a set of genes known collectively as the SOS response. Part of the SOS response includes genes that repair DNA damage, but another part of the response coordinates DNA replication and septation to prevent untimely cell division. The classic SOS gene product that inhibits cell division is SfiA (or SulA), which binds to FtsZ and prevents septum formation...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Bacterial Proteins
Share to Cell Division
Share to DNA, Bacterial

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to DNA Replication

Share to Escherichia coli

Share to Escherichia coli Proteins

Share to Gene Expression Regulation, Bacterial

Share to Mutation

Source

Open in PubMed Open full text

Identifiers

PMID: 9068638
DOI: 10.1128/jb.179.6.1931-1939.1997
PMCID: PMC178916

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

sfi-independent filamentation in Escherichia coli Is lexA dependent and requires DNA damage for induction.PMID 9068638Read ↗

A newly identified prophage-encoded gene, <i>ymfM</i> , causes SOS-inducible filamentation in <i>Escherichia coli</i>Semantic proximity Read ↗

Cell division, guillotining of dimer chromosomes and SOS induction in resolution mutants (dif, xerC and xerD) of Escherichia coli.Semantic proximity Read ↗

Role of SufI (FtsP) in cell division of Escherichia coli: evidence for its involvement in stabilizing the assembly of the divisome.Semantic proximity Read ↗

Roles of the recG gene product of Escherichia coli in recombination repair: effects of the delta recG mutation on cell division and chromosome partition.Semantic proximity Read ↗

Differential requirements of two recA mutants for constitutive SOS expression in Escherichia coli K-12.Semantic proximity Read ↗

Altered SOS induction associated with mutations in recF, recO and recR.Semantic proximity Read ↗

Homologous recombination-dependent initiation of DNA replication from DNA damage-inducible origins in Escherichia coli.Semantic proximity Read ↗

Genetic evidence for the requirement of RecA loading activity in SOS induction after UV irradiation in Escherichia coli.Semantic proximity Read ↗

Novel mechanism for UV sensitivity and apparent UV nonmutability of recA432 mutants: persistent LexA cleavage following SOS induction.Semantic proximity Read ↗

RecJ nuclease is required for SOS induction after introduction of a double-strand break in a RecA loading deficient recB mutant of Escherichia coli.Semantic proximity Read ↗

dif, a recA-independent recombination site in the terminus region of the chromosome of Escherichia coli.Semantic proximity Read ↗

A new mutation in Escherichia coli K12, isfA, which is responsible for inhibition of SOS functions.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.