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Article

Glutathione S-transferase M1 null genotype as a risk modifier for solvent-induced chronic toxic encephalopathy.

Scandinavian journal of work, environment & health - 1 Oct 1996

Söderkvist P, Ahmadi A, Akerbäck A, Axelson O, Flodin U

Abstract excerpt

OBJECTIVES: Exposure to organic solvents increases the risk of neuropsychiatric disability or chronic toxic encephalopathy (CTE). Polymorphisms in the biotransformation of xenobiotics and solvents may influence individual susceptibility to develop toxic effects. In this study the problem of whether there could be any association between the glutathione S-transferase M1 (GSTM1) null genotype and the risk for CTE,...

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Source

Identifiers

PMID: 8923609
DOI: 10.5271/sjweh.154

Genes

GSTM1

Provenance

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Related research

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Research trail→

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Glutathione S-transferase M1 null genotype as a risk modifier for solvent-induced chronic toxic encephalopathy.PMID 8923609Read ↗

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[The role of smoking and metabolic enzyme polymorphisms in the organic solvent induced chronic encephalopathy].Semantic proximity Read ↗

Glutathione S-transferase m1 and t1 null genotypes increase susceptibility to idiosyncratic drug-induced liver injury.Semantic proximity Read ↗

The Upper Midwest Health Study: gliomas and occupational exposure to chlorinated solvents.Semantic proximity Read ↗

Unveiling the etiological impact of GST-M1, GST-T1, and P53 genotypic variations on brain carcinogenesis.Semantic proximity Read ↗

Polymorphism of Glutathione S-transferase Genes and the Risk of Toxic Liver Damage in Petrochemical Workers.Semantic proximity Read ↗

Genetic polymorphisms in GSTM1, -P1, -T1, and CYP2E1 and the risk of adult brain tumors.Semantic proximity Read ↗

Polymorphisms of the glutathione S-transferases mu-1 (GSTM1) and theta-1 (GSTT1) and the risk of advanced alcoholic liver disease.Semantic proximity Read ↗

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