Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Susceptibility to the development of gastric mucosal damage in OLETF rat not expressing the CCK-A receptor.

Biochemical and biophysical research communications - 25 Jul 1996

Okumura T, Shoji E, Onodera S, Kohgo Y

Abstract excerpt

Otsuka Long-Evans Tokushima Fatty (OLETF) rats are a new model of a congenital defect of the CCK-A receptor gene and should be useful for determining CCK-A receptor function. Since the CCK-A receptor plays an important role in gastrointestinal function, in the present study we examined, using OLETF rats, the hypothesis that a defect of the CCK-A receptor may influence gastric mucosal integrity. The gastric...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Ethanol
Share to Gastric Mucosa
Share to Genetic Predisposition to Disease

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Indomethacin

Share to Rats, Inbred Strains

Source

Identifiers

PMID: 8713114
DOI: 10.1006/bbrc.1996.1091

Genes

CCK

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 8 closest

Susceptibility to the development of gastric mucosal damage in OLETF rat not expressing the CCK-A receptor.PMID 8713114Read ↗

Regulation of pancreatic exocrine function in Otsuka Long-Evans Tokushima Fatty (OLETF) rats without gene expression of cholecystokinin-A receptor.Semantic proximity Read ↗

Apoptosis in the pancreas of genetically diabetic rats with a disrupted cholecystokinin (CCK-A) receptor gene.Semantic proximity Read ↗

Decrease in exploratory behavior in naturally occurring cholecystokinin (CCK)-A receptor gene knockout rats.Semantic proximity Read ↗

Absence of the cholecystokinin-A receptor deteriorates homeostasis of body temperature in response to changes in ambient temperature.Semantic proximity Read ↗

Cholecystokinin-8 increases Fos-like immunoreactivity in the brainstem and myenteric neurons of rats through CCK1 receptors.Semantic proximity Read ↗

Normal feeding and body weight in Fischer 344 rats lacking the cholecystokinin-1 receptor gene.Semantic proximity Read ↗

A disrupted cholecystokinin A receptor gene induces diabetes in obese rats synergistically with ODB1 gene.Semantic proximity Read ↗

Mutated G-protein-coupled receptor GPR10 is responsible for the hyperphagia/dyslipidaemia/obesity locus of Dmo1 in the OLETF rat.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.