Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Metabolic disruptions in the adipocyte-hepatocyte fatty acid axis as causes of HyperapoB.

International journal of obesity and related metabolic disorders : journal of the International Association for the Study of Obesity - 1 May 1995

Sniderman A D, Cianflone K

Abstract excerpt

HyperapoB is the atherogenic dyslipoproteinemia characterized by increased numbers of LDL particles in plasma due to increased secretion of B100 lipoprotein particles by the liver. The lipid phenotype in affected patients is variable but an increased plasma apoB points to the increased LDL particle number. The adipocyte-hepatocyte fatty acid axis refers to the traffic in fatty acids from adipocytes to hepatocytes...

Topics

Adipocytes
Apolipoproteins B
Fatty Acids
Humans
Hyperlipoproteinemias
Lipoproteins
Liver
Phenotype
Triglycerides

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 7550535

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 29 Aug 2026. Prepared record publication-7550535-2026-08-29.1. Manifest publication-graph-cohort-2026-08-29.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 29 Aug 2026.

Community interpretation

Discuss the evidence

Ask a question, add context or challenge an interpretation. Community activity is separate from scientific evidence strength.

0

discussions

0

replies

Citations encouraged

Claims remain attributable.

No discussion yet

Be the first to add a careful, cited interpretation.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Metabolic disruptions in the adipocyte-hepatocyte fatty acid axis as causes of HyperapoB.PMID 7550535Read ↗

Abetalipoproteinemia and homozygous hypobetalipoproteinemia: a framework for diagnosis and management.Semantic proximity Read ↗

Pathophysiology of fatty acid oxidation disorders.Semantic proximity Read ↗

[Adipokine genetics: unbalanced protein secretion by human adipose tissue as a cause of the metabolic syndrome].Semantic proximity Read ↗

Lipid disorders and mutations in the APOB gene.Semantic proximity Read ↗

A fresh look at NASH pathogenesis. Part 1: the metabolic movers.Semantic proximity Read ↗

Biochemical aspects of X-linked adrenoleukodystrophy.Semantic proximity Read ↗

Substrate-Specific Function of PNPLA3 Facilitates Hepatic VLDL-Triglyceride Secretion During Stimulated LipogenesisSemantic proximity Read ↗

Secondary hypertriglyceridemia in children and adolescents.Semantic proximity Read ↗

Monogenic forms of insulin resistance: apertures that expose the common metabolic syndrome.Semantic proximity Read ↗

Monogenic hypocholesterolaemic lipid disorders and apolipoprotein B metabolism.Semantic proximity Read ↗

PNPLA3-I148M is a Neomorph that Interferes with Two Primary Hepatic Triglyceride Clearance PathwaysSemantic proximity Read ↗

Fatty pancreas: an initial indicator of ectopic fat deposition and metabolic syndromeSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.