Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Stroke in Pregnancy Brings Epigenetic Changes in Correlation with Affected Mitochondrial Dynamics and Inflammasome Mediated Apoptosis in Rodents

2 May 2025

Abstract excerpt

BACKGROUND: Pregnancy may be a risk factor for stroke in females. Stroke in pregnancy influences mitochondrial dynamics as well as the inflammatory responses in mothers. However, limited studies are available that report any epigenetic changes in the offspring following a stroke in mothers. In the present study we investigate the effect of stroke in pregnancy as a possible epigenetic modifier correlated with...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Neuroinflammation and Neurodegeneration Mechanisms
Share to Neurological Complications and Syndromes
Share to Pregnancy and preeclampsia studies

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 40314367
DOI: 10.1161/jaha.124.039411

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 3 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Stroke in Pregnancy Brings Epigenetic Changes in Correlation with Affected Mitochondrial Dynamics and Inflammasome Mediated Apoptosis in RodentsPMID 40314367Read ↗

The Role of Mitochondrial Dysfunction in Preeclampsia: Causative Factor or Collateral Damage?Semantic proximity Read ↗

Cerebral infarctions in the fetus and neonate: maternal-placental-fetal considerations.Semantic proximity Read ↗

Sex differences in the effects of PARP inhibition on microglial phenotypes following neonatal stroke.Semantic proximity Read ↗

Pandora's Box: mitochondrial defects in ischaemic heart disease and stroke.Semantic proximity Read ↗

Postpartum Plasma-Derived Exosomes Confer Mitochondrial Stabilization and Neuroprotection against Ischemic StrokeSemantic proximity Read ↗

Maternal Inflammation During Pregnancy and Offspring Brain Development: The Role of MitochondriaSemantic proximity Read ↗

Human umbilical mesenchymal stem cell-derived mitochondria ameliorate maternal phenotype by improving placental mitochondria and vascular function in angiotensin II-induced preeclampsia ratSemantic proximity Read ↗

Neonatal Encephalopathy is a Complex Phenotype Representing Reproductive and Pregnancy Exposome Effects on the Maternal-Placental-Fetal Triad.Semantic proximity Read ↗

Intergenerational Conditioning via Intermittent Parental Hypoxia Confers Stroke Resilience in OffspringSemantic proximity Read ↗

Is Mitochondrial Oxidative Stress a Viable Therapeutic Target in Preeclampsia?Semantic proximity Read ↗

Restoration of Miro1’s N-terminal GTPase function alleviates prenatal stress-induced mitochondrial fission via Drp1 modulationSemantic proximity Read ↗

Sex Differences in Mechanisms and Outcome of Neonatal Hypoxia-Ischemia in Rodent Models: Implications for Sex-Specific Neuroprotection in Clinical Neonatal PracticeSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.