Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Deletion of CD38 mitigates the severity of NEC in experimental settings by modulating macrophage-mediated inflammation

19 Sept 2024

Abstract excerpt

Necrotizing enterocolitis (NEC) is a form of potentially lethal gastrointestinal inflammation that primarily affects preterm neonates. It is crucial to recognize that, while the disease carries significant risks, timely and effective medical intervention can greatly enhance the chances of survival. Additionally, NEC is closely linked to the activation of macrophages, highlighting the complex interplay between the...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adenosine and Purinergic Signaling
Share to Autoimmune Neurological Disorders and Treatments
Share to Calcium signaling and nucleotide metabolism

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 39366069
DOI: 10.1016/j.redox.2024.103336

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 3 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Deletion of CD38 mitigates the severity of NEC in experimental settings by modulating macrophage-mediated inflammationPMID 39366069Read ↗

Regulation of intracellular levels of NAD: a novel role for CD38.Semantic proximity Read ↗

A Novel Role for Necroptosis in the Pathogenesis of Necrotizing EnterocolitisSemantic proximity Read ↗

New insights into necrotizing enterocolitis: From laboratory observation to personalized prevention and treatment.Semantic proximity Read ↗

The immunological landscape in necrotising enterocolitis.Semantic proximity Read ↗

Interactions between NAD+ metabolism and immune cell infiltration in ulcerative colitis: subtype identification and development of novel diagnostic modelsSemantic proximity Read ↗

Unveiling the role of NAD glycohydrolase CD38 in aging and age-related diseases: insights from bibliometric analysis and comprehensive reviewSemantic proximity Read ↗

Gene expression profiling in necrotizing enterocolitis reveals pathways common to those reported in Crohn’s diseaseSemantic proximity Read ↗

Characterization of the pathoimmunology of necrotizing enterocolitis reveals novel therapeutic opportunitiesSemantic proximity Read ↗

Necrotizing enterocolitis: recent scientific advances in pathophysiology and prevention.Semantic proximity Read ↗

Circulating eNAMPT as a Diagnostic Biomarker in the Critically Ill: Acute Pancreatitis, Sepsis, Trauma, and Acute Respiratory Distress SyndromeSemantic proximity Read ↗

CD38-NADase is a new major contributor to Duchenne muscular dystrophic phenotype.Semantic proximity Read ↗

CD38 ecto-enzyme in immune cells is induced during aging and regulates NAD+ and NMN levels.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.