Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

A Statistical Testing Strategy Accounting for Random and Nonrandom (Skewed) X-Chromosome Inactivation Identifies Lung Cancer Susceptibility Loci among Smokers.

Human heredity - 1 Jan 2024

Jantzen Rodolphe, Camilleri-Broët Sophie, Ezer Nicole, Broët Philippe

Abstract excerpt

INTRODUCTION: Lung cancer is the most common cancer worldwide in mortality and the second in incidence. Epidemiological studies found a higher lung cancer risk for smoking women in comparison to men, but these sex differences, irrespective of smoking habits, remain controversial. One of the hypotheses concerns the genetic contribution of the sex chromosomes. However, while genome-wide association studies...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Chromosomes, Human, X
Share to Computer Simulation

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Genetic Loci

Share to Genome-Wide Association Study

Share to Lung Neoplasms

Share to Smokers

Share to Smoking

Share to X Chromosome Inactivation

Source

Identifiers

PMID: 38880085
DOI: 10.1159/000539520

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

A Statistical Testing Strategy Accounting for Random and Nonrandom (Skewed) X-Chromosome Inactivation Identifies Lung Cancer Susceptibility Loci among Smokers.PMID 38880085Read ↗

No genetic causal association between human papillomavirus and lung cancer risk: a bidirectional two-sample Mendelian randomization analysis.Semantic proximity Read ↗

Genome-wide interaction analysis identified low-frequency variants with sex disparity in lung cancer risk.Semantic proximity Read ↗

X Chromosome Associations with Chronic Obstructive Pulmonary Disease and Related Phenotypes: An X Chromosome-wide Association StudySemantic proximity Read ↗

X chromosome associations with chronic obstructive pulmonary disease and related phenotypes: an X chromosome-wide association study.Semantic proximity Read ↗

Chapter 6: Lung cancer in never smokers: epidemiology and risk prediction models.Semantic proximity Read ↗

Genetic susceptibility to lung cancer based on candidate genes in a sample from the Mexican Mestizo population: a case-control study.Semantic proximity Read ↗

Risk mapping of lung cancer: a comprehensive appraisal of published meta-analyses incorporating Mendelian randomization studies.Semantic proximity Read ↗

The association between human papillomavirus and lung cancer: A Mendelian randomization study.Semantic proximity Read ↗

No Genetic Causal Association Between Human Papillomavirus and Lung Cancer Risk A Bidirectional Two-Sample Mendelian Randomization AnalysisSemantic proximity Read ↗

FARVATX: Family-Based Rare Variant Association Test for X-Linked Genes.Semantic proximity Read ↗

A causal association between Epstein-Barr virus infection and lung cancer: A two-sample Mendelian Randomization study.Semantic proximity Read ↗

A causal association between chemokines and the risk of lung cancer: a univariate and multivariate mendelian randomization study.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.