Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The lncRNA Snhg11, a new candidate contributing to neurogenesis, plasticity, and memory deficits in Down syndrome

26 Feb 2024

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer-related molecular mechanisms research
Share to Down syndrome and intellectual disability research
Share to RNA modifications and cancer

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 38409595
DOI: 10.1038/s41380-024-02440-9

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 1 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The lncRNA Snhg11, a new candidate contributing to neurogenesis, plasticity, and memory deficits in Down syndromePMID 38409595Read ↗

The lncRNA Snhg11 is required for synaptic function, neurogenesis and memory and is downregulated in the dentate gyrus of Down syndrome mouse modelsSemantic proximity Read ↗

The lncRNA Snhg11, a new candidate contributing to neurogenesis, plasticity and memory deficits in Down syndromeSemantic proximity Read ↗

Epigenetics: The neglected key to minimize learning and memory deficits in Down syndromeSemantic proximity Read ↗

Integrative multi-omic analysis reveals conserved cell-projection deficits in human Down syndrome brainsSemantic proximity Read ↗

Down syndrome: Neurobiological alterations and therapeutic targetsSemantic proximity Read ↗

A single cell multi-omic analysis identifies molecular and gene-regulatory mechanisms dysregulated in the developing Down syndrome neocortexSemantic proximity Read ↗

Neurodevelopment in Down syndrome: Concordance in humans and modelsSemantic proximity Read ↗

A single-cell multiomic analysis identifies molecular and gene-regulatory mechanisms dysregulated in developing Down syndrome neocortexSemantic proximity Read ↗

Single nucleus RNA-seq in the hippocampus of a Down syndrome mouse model reveals new key players in memorySemantic proximity Read ↗

Down Syndrome Developmental Brain Transcriptome Reveals Defective Oligodendrocyte Differentiation and MyelinationSemantic proximity Read ↗

REST Targets JAK–STAT and HIF-1 Signaling Pathways in Human Down Syndrome Brain and Neural CellsSemantic proximity Read ↗

Analysis of Adult Cerebral Cortex and Hippocampus Transcriptomes Reveals Unique Molecular Changes in the Ts1Cje Mouse Model of D own SyndromeSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.