Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Contribution of Complement, Microangiopathy and Inflammation in Idiopathic Inflammatory Myopathies

20 Dec 2023

Abstract excerpt

PURPOSE OF REVIEW: Idiopathic inflammatory myopathies (IIMs) are a heterogeneous group characterized by muscle weakness and skin symptoms and are categorized into six subtypes: dermatomyositis (DM), polymyositis (PM), anti-synthetase syndrome (ASS), immune-mediated myopathy (IMNM), inclusion body myopathy (IBM), and overlap myositis. Myositis-specific autoantibodies were detected for the diagnosis and...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Inflammatory Myopathies and Dermatomyositis
Share to Myasthenia Gravis and Thymoma
Share to Spondyloarthritis Studies and Treatments

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 38143369
DOI: 10.3233/jnd-230168

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 23 Oct 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Contribution of Complement, Microangiopathy and Inflammation in Idiopathic Inflammatory MyopathiesPMID 38143369Read ↗

Pathogenic aspects of dermatomyositis, polymyositis and overlap myositis.Semantic proximity Read ↗

Idiopathic inflammatory myopathies: current trends in pathogenesis, clinical features, and up-to-date treatment recommendations.Semantic proximity Read ↗

Complement and MHC patterns can provide the diagnostic framework for inflammatory neuromuscular diseasesSemantic proximity Read ↗

Chemokine Profile of Different Inflammatory Myopathies Reflects Humoral versus Cytotoxic Immune ResponsesSemantic proximity Read ↗

Deciphering the clinical presentations, pathogenesis, and treatment of the idiopathic inflammatory myopathies.Semantic proximity Read ↗

Low copy numbers of complement C4 and C4A deficiency are risk factors for myositis, its subgroups and autoantibodies.Semantic proximity Read ↗

An update on pathogenic mechanisms of inflammatory myopathiesSemantic proximity Read ↗

Clinical-pathologic classification of anti-HMGCR-positive immune-mediated necrotizing myopathySemantic proximity Read ↗

Classification of myositis.Semantic proximity Read ↗

Plasma proteome reflects tissue damage and clinical manifestations in patients with inflammatory myopathiesSemantic proximity Read ↗

The pathogenesis of anti-signal recognition particle necrotizing myopathy: A ReviewSemantic proximity Read ↗

Distinct Cytokine and Cytokine Receptor Expression Patterns Characterize Different Forms of MyositisSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.