Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Oxidative Stress Contributes to Inflammatory and Cellular Damage in Systemic Lupus Erythematosus: Cellular Markers and Molecular Mechanism

1 Feb 2023

Abstract excerpt

Systemic lupus erythematosus (SLE) is a multifactorial autoimmune disease with complex pathogenesis, the treatment of which relies exclusively on the use of immunosuppressants. Increased oxidative stress is involved in causing inflammatory and cellular defects in the pathogenesis of SLE. Various inflammatory and cellular markers including oxidative modifications of proteins, lipids, and DNA contribute to immune...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Atherosclerosis and Cardiovascular Diseases
Share to Neutrophil, Myeloperoxidase and Oxidative Mechanisms
Share to Systemic Lupus Erythematosus Research

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 36761905
DOI: 10.2147/jir.s399284

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Oxidative Stress Contributes to Inflammatory and Cellular Damage in Systemic Lupus Erythematosus: Cellular Markers and Molecular MechanismPMID 36761905Read ↗

The Role of the Oxidative State and Innate Immunity Mediated by TLR7 and TLR9 in Lupus NephritisSemantic proximity Read ↗

Immunometabolism in the pathogenesis of systemic lupus erythematosus: an updateSemantic proximity Read ↗

Oxidative Stress in Autoimmune Diseases: An Under Dealt Malice.Semantic proximity Read ↗

The Role of Immunometabolism in the Pathogenesis of Systemic Lupus ErythematosusSemantic proximity Read ↗

The Real Culprit in Systemic Lupus Erythematosus: Abnormal Epigenetic RegulationSemantic proximity Read ↗

Mitochondrial aberrations in systemic lupus erythematosus pathogenesis: Insights and therapeutic implicationsSemantic proximity Read ↗

Mitochondrial impairment and repair in the pathogenesis of systemic lupus erythematosusSemantic proximity Read ↗

Molecular and Cellular Bases of Immunosenescence, Inflammation, and Cardiovascular Complications Mimicking “Inflammaging” in Patients with Systemic Lupus ErythematosusSemantic proximity Read ↗

Immune cell multiomics analysis reveals contribution of oxidative phosphorylation to B-cell functions and organ damage of lupusSemantic proximity Read ↗

Mitochondria as a key player in systemic lupus erythematosusSemantic proximity Read ↗

Machine learning combined multi-omics analysis to explore key oxidative stress features in systemic lupus erythematosusSemantic proximity Read ↗

Molecular and Cellular Mechanisms of Systemic Autoimmune Diseases: Lessons Learned from Human Genetic Studies and Therapies in Systemic Lupus Erythematosus (SLE)Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.