Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

LINE-1 and Alu methylation signatures in autism spectrum disorder and their associations with the expression of autism-related genes

17 Aug 2022

Abstract excerpt

Long interspersed nucleotide element-1 (LINE-1) and Alu elements are retrotransposons whose abilities cause abnormal gene expression and genomic instability. Several studies have focused on DNA methylation profiling of gene regions, but the locus-specific methylation of LINE-1 and Alu elements has not been identified in autism spectrum disorder (ASD). Here we interrogated locus- and family-specific methylation...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Autism Spectrum Disorder Research
Share to Chromosomal and Genetic Variations
Share to Virology and Viral Diseases

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 35978033
DOI: 10.1038/s41598-022-18232-6

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

LINE-1 and Alu methylation signatures in autism spectrum disorder and their associations with the expression of autism-related genesPMID 35978033Read ↗

Exploratory Analysis of L1 retrotransposons expression in AutismSemantic proximity Read ↗

Whole-genome analysis of <i>de novo</i> and polymorphic retrotransposon insertions in Autism Spectrum DisorderSemantic proximity Read ↗

Genome-wide DNA methylation profiling identifies convergent molecular signatures associated with idiopathic and syndromic forms of autism in post-mortem human brain tissueSemantic proximity Read ↗

Short tandem repeat expansions in cortical layer-specific genes implicate in phenotypic severity and adaptability of autism spectrum disorder.Semantic proximity Read ↗

A Single-Cell Atlas of DNA Methylation in Autism Spectrum Disorder Reveals Distinct Regulatory and Aging SignaturesSemantic proximity Read ↗

Genome-wide patterns of <i>de novo</i> tandem repeat mutations and their contribution to autism spectrum disordersSemantic proximity Read ↗

Noncoding <i>de novo</i> mutations contribute to autism spectrum disorder via chromatin interactionsSemantic proximity Read ↗

Non-coding de novo mutations in chromatin interactions are implicated in autism spectrum disorder.Semantic proximity Read ↗

Genetic and Epigenetic Etiology Underlying Autism Spectrum DisorderSemantic proximity Read ↗

Genomic architecture of autism from comprehensive whole-genome sequence annotation.Semantic proximity Read ↗

Merging data from genetic and epigenetic approaches to better understand autistic spectrum disorder.Semantic proximity Read ↗

Peripheral blood DNA methylation and autism spectrum disorderSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.